- ►endojournals.org AM Vincent, L Perrone, KA Sullivan, C Backus … - Endocrinology, 2007 - Endocrine Soc The receptor for advanced glycation end products (RAGE) may promote diabetic
vascular and renal disease through the activation of intracellular signaling
pathways that promote oxidative stress. Oxidative stress is a mediator of ... Cited by 39 - Related articles - BL Direct - All 6 versions
M Soccio, E Toniato, V Evangelista, M … - European journal of clinical investigation, 2005 - pt.wkhealth.com Several risk factors for coronary artery disease (CAD) induce atherosclerosis
through endothelial activation and dysfunction, and ample evidence now suggests
that the balance between production and removal of reactive oxygen species ... Cited by 33 - Related articles - All 7 versions
KG Ewens, RA George, K Sharma, FN … - Diabetes, 2005 - Am Diabetes Assoc Several lines of evidence, including familial aggregation, suggest that allelic
variation contributes to risk of diabetic nephropathy. To assess the evidence
for specific susceptibility genes, we used the transmission/disequilibrium ... Cited by 34 - Related articles - BL Direct - All 5 versions
LJN Jensen, J Østergaard, A Flyvbjerg - Horm. Metab. Res., 2005 - 万方数据资源系统 At present, diabetic kidney disease affects about 15 - 25 % of all type 1
diabetic patients and 20 - 40 % of all patients with type 2 diabetes. The
mechanisms underlying the development of diabetic kidney disease are ... Cited by 27 - Related articles - All 6 versions
- ►diabetesjournals.org V Thallas-Bonke, SR Thorpe, MT Coughlan, K … - Diabetes, 2008 - Am Diabetes Assoc RESULTS—Apocynin attenuated diabetes-associated increases in albuminuria and
glomerulosclerosis. Circulating, renal cytosolic, and skin collagen–associated
AGE levels in diabetic rats were not reduced by apocynin. Diabetes-induced ... Cited by 24 - Related articles - BL Direct - All 4 versions
DPK Ng, AS Krolewski - Current Molecular Medicine, 2005 - ingentaconnect.com 1 Department of Community, Occupational and Family Medicine, and Centre for
Molecular Epidemiology, National University of Singapore 2 Joslin Diabetes
Center, and Department of Medicine, Harvard Medical School, Boston, MA, USA ... Cited by 22 - Related articles - BL Direct - All 3 versions
- ►googlepages.com [PDF] … , M Balasubramanyam, M Rema, V Mohan - Diabetic Medicine, 2006 - pt.wkhealth.com Aims: While the downstream effects of increased reactive oxygen species (ROS) in
the pathogenesis of diabetes were well studied, only a few studies have explored
the cellular sources of ROS. We examined whether protection against ... Cited by 15 - Related articles - BL Direct - All 7 versions
Q Lan, T Zheng, M Shen, Y Zhang, SS Wang, … - Human genetics, 2007 - Springer Abstract Oxidative damage caused by reactive oxy- gen species (ROS) and other
free radicals is involved in a number of pathological conditions including
cancer. In a population-based case-control study of non-Hodg- kin lymphoma ... Cited by 15 - Related articles - All 5 versions
T Ogawa, H Hirose, K Miyashita, I Saito, T … - Metabolism, 2005 - Elsevier GPR40 is a member of the G-protein–coupled receptors. Recent studies suggest
that GPR40 is highly expressed in pancreatic β cells and insulin-secreting cell
lines, and that fatty acids increase intracellular calcium concentration ... Cited by 15 - Related articles - All 9 versions
