Web Images Videos Maps News Shopping Gmail more »
Sign in
Scholar Home  
  Advanced Scholar Search
Scholar Preferences
Scholar Results 1 - 10 of about 27 citing Major: Interleukin-1β Stimulation of c-Jun NH2-Terminal Kinase Activity in Insulin-Secreting Cells. (0.08 sec) 

Inflammatory mediators and islet ß-cell failure: a link between type 1 and type 2 …


MY Donath, J Størling, K Maedler, T Mandrup- … - Journal of Molecular …, 2003 - Springer
Abstract Pancreatic islet β-cell death occurs in type 1 and 2 diabetes mellitus, leading to
absolute or relative insulin deficiency. β-cell death in type 1 diabetes is due predominantly to
autoimmunity. In type 2 diabetes ...
Cited by 145 - Related articles - BL Direct - All 3 versions

Oxidative stress induces nucleo-cytoplasmic translocation of pancreatic …


D Kawamori, Y Kajimoto, H Kaneto, Y Umayahara, Y … - Diabetes, 2003 - Am Diabetes Assoc
Oxidative stress is induced in pancreatic β-cells under diabetic conditions and causes β-cell
dysfunction. Antioxidant treatment of diabetic animals leads to recovery of insulin biosynthesis and
increases the expression of its ...
Cited by 68 - Related articles - BL Direct - All 4 versions

Intracellular stress signaling pathways activated during human islet preparation …


S Abdelli, J Ansite, R Roduit, T Borsello, I Matsumoto, T … - Diabetes, 2004 - Am Diabetes Assoc
Type 1 diabetes is an autoimmune disease resulting from a progressive decrease in β-cell mass and
function (1). The mediators of β-cell dysfunction and destruction include macrophages, T-cells and
their inflammatory products, such ...
Cited by 50 - Related articles - BL Direct - All 8 versions

Involvement of oxidative stress in the pathogenesis of diabetes


H Kaneto, N Katakami, D Kawamori, T … - Antioxidants & redox …, 2007 - liebertonline.com
F AILURE OF PANCREATIC cells is the common characteris- tic of type 1 and type 2 diabetes. Type 1
diabetes melli- tus is induced by destruction of pancreatic cells, which is mediated by an
autoimmune mechanism and the ...
Cited by 48 - Related articles - BL Direct - All 4 versions

Cross-talk between phosphatidylinositol 3-kinase/AKT and c-jun NH2-terminal …

- endojournals.org
R Aikin, D Maysinger, L Rosenberg - Endocrinology, 2004 - Endocrine Soc
Therapeutic strategies aimed at the inhibition of specific cell death mechanisms may increase islet
yield and improve cell viability and function after routine isolation. The aim of the current study
was to explore the possibility of ...
Cited by 42 - Related articles - BL Direct - All 3 versions

Reversal of diet-induced insulin resistance with a single bout of exercise in the rat: …


ER Ropelle, JR Pauli, PO Prada, CT de … - The Journal of …, 2006 - Physiological Soc
Lifestyle interventions including exercise programmes are cornerstones in the prevention of
obesity-related diabetes. In this study, we demonstrate that a single bout of exercise inhibits
high-fat diet-induced insulin ...
Cited by 35 - Related articles - BL Direct - All 8 versions

Western diet modulates insulin signaling, c-Jun N-terminal kinase activity, and …

- endojournals.org
PO Prada, HG Zecchin, AL Gasparetti, MA Torsoni, M … - Endocrinology, 2005 - Endocrine Soc
The mechanisms by which diet-induced obesity is associated with insulin resistance are not well
established, and no study has until now integrated, in a temporal manner, functional insulin action
data with insulin signaling in key ...
Cited by 30 - Related articles - All 5 versions

[PDF] Cardiovascular Diabetology


EZ Fisman, A Tenenbaum, M Motro - Cardiovascular diabetology, 2003 - biomedcentral.com
Address: 1Sackler Faculty of Medicine, Tel-Aviv University, 69978 Ramat-Aviv, Tel-Aviv, Israel,
2Cardiovascular Diabetology Research Foundation, 58484 Holon, Israel and 3Cardiac Rehabilitation
Institute, Sheba Medical Center, ...
Cited by 32 - Related articles - View as HTML - All 15 versions

The JNK binding domain of islet-brain 1 inhibits IL-1 induced JNK activity and …


MA Nikulina, N Sandhu, Z Shamim, NA Andersen, A … - Cytokine, 2003 - Elsevier
The stress-activated protein kinase c-Jun NH 2 -terminal kinase (JNK) is a central signal for
interleukin-1β (IL-1β)-induced apoptosis in insulin-producing β-cells. The cell-permeable peptide
inhibitor of JNK (JNKI1), that ...
Cited by 20 - Related articles - All 3 versions

Molecular mechanisms and clinical implications of reversible protein S- …


JJ Mieyal, MM Gallogly, S Qanungo, EA … - Antioxidants & Redox …, 2008 - liebertonline.com
Page 1. 1941 1 Department of Pharmacology, Case Western Reserve University, Cleveland, Ohio. 2 Louis
B. Stokes Cleveland Veterans Affairs Medical Research Center, Cleveland, Ohio. ANTIOXIDANTS & REDOX
SIGNALING Volume 10, Number 11, 2008 © Mary Ann Liebert, Inc. DOI: 10.1089/ars.2008.2089 ...
Cited by 22 - Related articles - All 4 versions


Result Page: 

1

2

3

Next


 


Go to Google Home - About Google - About Google Scholar

©2009 Google