- ►rug.nl [PDF] AJ Smit, HL Lutgers - Current medicinal chemistry, 2004 - ingentaconnect.com Abstract: Advanced glycation endproducts (AGE) are a class of compounds
resulting from glycation and oxidation of proteins, lipids or nucleic acids.
Glycation is the non-enzymatic addition or insertion of saccharide ... Cited by 41 - Related articles - BL Direct - All 10 versions
- ►shouxi.net A Rosso, A Balsamo, R Gambino, P Dentelli, R … - Journal of Biological Chemistry, 2006 - ASBMB Adverse metabolic factors, including oxidized small and dense low density
lipoprotein (ox-dmLDL) can contribute to the reduced number and the impaired
functions of circulating endothelial progenitors (EPC) in diabetic ... Cited by 36 - Related articles - BL Direct - All 5 versions
- ►fasebj.org [PDF] MF Brizzi, P Dentelli, A Rosso, C Calvi, R … - The FASEB Journal, 2004 - FASEB The FASEB Journal express article 10.1096/fj.03-1053fje. Published online June
4, 2004. ... RAGE- and TGF-β receptor-mediated signals converge on ... STAT5
and p21 waf to control cell-cycle progression of ... Maria Felice Brizzi,* ... Cited by 20 - Related articles - BL Direct - All 4 versions
- ►nih.gov MP Cohen, FN Ziyadeh, S Chen - The Journal of laboratory and clinical medicine, 2006 - Elsevier Diabetes confers a four-fold risk for atherosclerotic cardiovascular disease,
manifest in the vessel wall as intimal thickening, smooth muscle hypertrophy,
lipid accumulation, extracellular matrix expansion, and inflammatory cell ... Cited by 11 - Related articles - All 6 versions
- ►jbc.org P Dentelli, A Rosso, A Zeoli, R Gambino, L … - Journal of Biological Chemistry, 2007 - ASBMB Lipid abnormalities and oxidative stress, by stimulating mesangial cell (MC)
proliferation, can contribute to the development of diabetes-associated renal
disease. In this study we investigated the molecular events elicited by ... Cited by 7 - Related articles - All 5 versions
R Gambino, B Uberti, N Alemanno, E Pisu, G … - Atherosclerosis, 2004 - Elsevier We aimed to determine if increased non-enzymatic glycosylation of the LDL was
sufficient to increase the susceptibility to in vivo oxidation of the LDL
particles. Twenty-two type 2 diabetic patients (11 males and 11 females) ... Cited by 7 - Related articles - All 7 versions
R Gambino, E Pisu, G Pagano, M Cassader - Lipids, 2006 - Springer ABSTRACT: Multifactorial etiology is involved in premature atherosclerosis
related to diabetes. Most of the mechanisms that are responsible for the
etiology in diabetes have remained un- solved so far. Type 1 diabetes is ... Cited by 5 - Related articles - BL Direct - All 4 versions
E Santini, R Lupi, S Baldi, S Madec, D … - Diabetologia, 2008 - Springer Abstract Aims/hypothesis Inflammation is a mechanism of glomeru- lar damage in
chronic glomerulopathies. LDL may increase the production of inflammatory
cytokines in renal tissues. However, the relative role of native, oxidised ... Cited by 3 - Related articles - All 3 versions
S Braschi, M Geoffrion, A Nguyen, Y … - Diabetologia, 2006 - Springer Abstract Aims/hypothesis: When LDLs are exposed to glucose in vitro, glycation
of apolipoprotein B100 (apoB) leads to a loss in its affinity for the LDL
receptor and reproducible alterations in the immunoreactivity of specific ... Cited by 2 - Related articles - BL Direct - All 3 versions
