EC Tsilibary - The Journal of Pathology, 2003 - ingentaconnect.com The alterations in the microvascular system of diabetes mellitus patients are
responsible for the most devastating complications of this widespread disease.
In the kidney, the microan- giopathy leads to thickening of the glomerular ... Cited by 129 - Related articles - BL Direct - All 7 versions
- ►amjpathol.org JH Li, W Wang, XR Huang, M Oldfield, AM … - American Journal of Pathology, 2004 - ASIP Advanced glycation end products (AGEs) have been shown to play a role in tubular
epithelial-myofibroblast transdifferentiation (TEMT) in diabetic nephropathy,
but the intracellular signaling pathway remains unknown. We report here ... Cited by 99 - Related articles - BL Direct - All 5 versions
S Mezzano, A Droguett, ME Burgos, LG Ardiles … - Kidney International, 2003 - nature.com Angiotensin-converting enzyme (ACE) immunostaining was elevated in tubular cells
and appeared in interstitial cells. Elevated levels of angiotensin II (Ang II)
immunostaining were observed in tubular and infiltrating interstitial ... Cited by 86 - Related articles - All 4 versions
- ►physiology.org [PDF] JM Bohlender, S Franke, G Stein, G Wolf - American Journal of Physiology- Renal Physiology, 2005 - Am Physiological Soc 173 articles, 82 of which you can access free at: This article cites
http://ajprenal.physiology.org/cgi/content/full/289/4/F645#BIBL ... 11 other
HighWire hosted articles, the first 5 are: This article has been cited by Cited by 80 - Related articles - BL Direct - All 7 versions
T Chavakis, A Bierhaus, PP Nawroth - Microbes and infection, 2004 - Elsevier The present review focuses on the role of RAGE, the “receptor for advanced
glycation end products”, in the inflammatory response. RAGE is a multiligand
receptor that propagates cellular dysfunction in several inflammatory ... Cited by 73 - Related articles - All 4 versions
KV Ramana, B Friedrich, S Srivastava, A … - Diabetes, 2004 - Am Diabetes Assoc Activation of the polyol pathway has been linked to the development of secondary
diabetic complications. However, the underlying molecular mechanisms remain
unclear. To probe the contribution of this pathway, we examined whether ... Cited by 61 - Related articles - BL Direct - All 4 versions
MO Chaney, WB Stine, TA Kokjohn, YM Kuo, … - BBA-Molecular Basis of Disease, 2005 - Elsevier In the AD brain, there are elevated amounts of soluble and insoluble Aβ
peptides which enhance the expression of membrane bound and soluble receptor for
advanced glycation end products (RAGE). The binding of soluble Aβ to ... Cited by 42 - Related articles - All 4 versions
AJ Jenkins, JD Best, RL Klein, TJ Lyons - Diabetes/Metabolism Reviews - interscience.wiley.com The chronic vascular complications of diabetes (nephropathy, retinopathy and
accelerated atherosclerosis) are a major cause of morbidity and premature
mortality. In spite of the more widespread availability of intensive ... Cited by 36 - Related articles - BL Direct - All 4 versions
MC Thomas, JM Forbes, ME Cooper - American journal of therapeutics, 2005 - journals.lww.com Chronic hyperglycemia and oxidative stress in diabetes results in the formation
and accumulation advanced glycation end products (AGEs). AGEs have a wide range
of chemical, cellular, and tissue effects that contribute to the ... Cited by 37 - Related articles - BL Direct - All 7 versions
K Susztak, E Ciccone, P McCue, K Sharma, EP … - PLoS Medicine, 2005 - medicine.plosjournals.org We found that expression of scavenger receptor CD36 coincided with proximal
tubular epithelial cell (PTEC) apoptosis and TED specifically in human DNP. High
glucose stimulated cell surface expression of CD36 in PTECs. CD36 ... Cited by 36 - Related articles - Cached - BL Direct - All 19 versions
