E Sobngwi, P Boudou, F Mauvais-Jarvis, H … - The Lancet, 2003 - Elsevier There was no difference between the groups with respect to percent body fat and
insulin sensitivity. Five of the 15 exposed participants, but none of the
controls had impaired glucose tolerance (p=0ยท02). Early insulin secretion ... Cited by 100 - Related articles - All 8 versions
C Gallou-Kabani, C Junien - Diabetes, 2005 - Am Diabetes Assoc Human epidemiological studies and appropriately designed dietary interventions
in animal models have provided considerable evidence to suggest that maternal
nutritional imbalance and metabolic disturbances, during critical time ... Cited by 96 - Related articles - All 10 versions
DA Rees, JC Alcolado - Diabetic Medicine, 2005 - cms1.daegu.ac.kr Animal experimentation has a long history in the field of dia- betes research.
The aim of this article is to review the commonly used animal models and discuss
the recent technological advances that are being employed in the ... Cited by 97 - Related articles - View as HTML - All 7 versions
- ►ucilmsa.org [PDF] DS Fernandez-Twinn, SE Ozanne - Physiology & behavior, 2006 - Elsevier Fetal programming is gaining momentum as a highly documented phenomenon which
links poor early growth to adult disease. It is backed up by large cohorts in
epidemiological studies worldwide and has been tested in various animal ... Cited by 76 - Related articles - All 5 versions
- ►endojournals.org M Manikkam, EJ Crespi, DD Doop, C … - Endocrinology, 2004 - Endocrine Soc Alterations in the maternal endocrine, nutritional, and metabolic environment
disrupt the developmental trajectory of the fetus, leading to adult diseases.
Female offspring of rats, subhuman primates, and sheep treated prenatally ... Cited by 55 - Related articles - BL Direct - All 6 versions
SK Karnik, H Chen, GW McLean, JJ Heit, X … - Science, 2007 - sciencemag.org A list of selected additional articles on the Science Web sites ... 4
article(s) on the ISI Web of Science. cited by This article has been ... :
subject collections This article appears in the following Cited by 56 - Related articles - BL Direct - All 8 versions
PD Gluckman, MA Hanson, AS Beedle - Bioessays, 2007 - interscience.wiley.com That there is a heritable or familial component of susceptibility to chronic
non-communicable diseases such as type 2 diabetes, obesity and cardiovascular
disease is well established, but there is increasing evidence that some ... Cited by 55 - Related articles - BL Direct - All 4 versions
B Portha - Diabetes/Metabolism Reviews - interscience.wiley.com Now that the reduction in -mass has been clearly established in humans with type
2 diabetes mellitus (T2DM) [1-4], the debate focuses on the possible mechanisms
responsible for decreased -cell number and impaired -cell function and ... Cited by 50 - Related articles - BL Direct - All 4 versions
PW Franks, HC Looker, S Kobes, L Touger, … - Diabetes, 2006 - Am Diabetes Assoc The in utero environment is a powerful risk factor for type 2 diabetes in
offspring, but little is known about the risk conveyed by nondiabetic
gestational glucose levels. This issue was explored in 911 nondiabetic Pima ... Cited by 50 - Related articles - BL Direct - All 11 versions
- ►angrylapdog.com BE Levin - Philosophical Transactions of the Royal Society B: …, 2006 - rstb.royalsocietypublishing.org.p.angrylapdog.com Epidemiological studies in humans suggest that maternal undernutrition, obesity
and diabetes during gestation and lactation can all produce obesity in
offspring. Animal models have allowed us to investigate the independent ... Cited by 42 - Related articles - BL Direct - All 10 versions
