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Scholar Results 1 - 5 of 5 citing Özmen: Inhibition of Thrombin Abrogates the Instant Blood-Mediated Inflammatory Reaction.... (0.08 sec) 

Evidence for functional nicotinic receptors on pancreatic β cells


H Yoshikawa, E Hellström-Lindahl, V Grill - Metabolism, 2005 - Elsevier
Epidemiological studies associate smoking with reduced insulin secretion. We
hypothesized that nicotine could negatively affect pancreatic β-cell function.
Acute or 48-hour exposures to nicotine (10 −4 to 10 −6 mol/L) ...
Cited by 10 - Related articles - All 11 versions

Evidence that insulin secretion influences SNAP-25 through proteasomal activation


Z Ma, N Portwood, A Foss, V Grill, A Björklund - Biochemical and Biophysical Research …, 2005 - Elsevier
Regulation of SNARE proteins by glucose in pancreatic islets is complex and
insufficiently clarified. We aimed to study effects of glucose per se separate
from enhancing effects on exocytosis. A 24 h culture of rat islets at ...
Cited by 4 - Related articles - All 4 versions

Mechanism of action of the oral direct thrombin inhibitor ximelagatran


C Mattsson, TC Sarich, SC Carlsson - Seminars in Vascular Medicine, 2005 - thieme-connect.com
Thrombin plays a central role in thrombus formation through its conversion of
fibrinogen to fibrin and activation of platelets as well as amplifying its own
generation by feedback activation via factors V, VIII, and XI. ...
Cited by 3 - Related articles - BL Direct - All 3 versions

[PDF] Complement activation triggered by biomaterial surfaces


J Andersson - … Summaries of Uppsala Dissertations from the Faculty … - uu.diva-portal.org
Today there are a vast number of medical devices in temporary or permanent
contact with human tissues. Blood-biomaterial contact is known to trigger the
complement system and results in generation of fluid phase anaphylatoxins ...
Cited by 2 - Related articles - View as HTML

[PDF] Aspects on the modulation of potassium channels in insulin-producing β-cells


Z Ma - diss.kib.ki.se
This thesis attempts to further clarify mechanisms behind the negative effects
of over- stimulation and the beneficial effect of ß-cell rest. For this purpose
diazoxide - opener of K+-ATP channels, which reversibly inhibits ...
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