- ►nih.gov L Myatt - The Journal of Physiology, 2006 - Physiological Soc Fetal programming occurs when the normal pattern of fetal development is
disrupted by an abnormal stimulus or 'insult' applied at a critical point in in
utero development. This then leads to an effect, for example diabetes or ... Cited by 65 - Related articles - BL Direct - All 8 versions
- ►endojournals.org N Jansson, SL Greenwood, BR Johansson, TL … - Journal of Clinical Endocrinology & Metabolism, 2003 - Endocrine Soc The activity and expression of placental nutrient transporters are primary
determinants for the supply of nutrients to the fetus, and these nutrients in
turn regulate fetal growth. We developed an experimental system to assess ... Cited by 52 - Related articles - BL Direct - All 6 versions
- ►endojournals.org AL Magnusson, IJ Waterman, M Wennergren, … - Journal of Clinical Endocrinology & Metabolism, 2004 - Endocrine Soc Triglyceride (TG) hydrolases in the placental microvillous plasma membrane (MVM)
release fatty acids from circulating lipoproteins and represent the critical
initial step in transplacental fatty acid transfer. We investigated the ... Cited by 40 - Related articles - BL Direct - All 5 versions
- ►nih.gov N Jansson, J Pettersson, A Haafiz, A … - The Journal of Physiology, 2006 - Physiological Soc Intrauterine growth restriction (IUGR) represents an important risk factor for
perinatal complications and for adult disease. IUGR is associated with a
down-regulation of placental amino acid transporters; however, whether ... Cited by 36 - Related articles - BL Direct - All 9 versions
T Jansson, T Powell - Clinical Science, 2007 - clinsci.org Adverse influences during fetal life alter the structure and function of
distinct cells, organ systems or homoeostatic pathways, thereby 'programming'
the individual for an increased risk of developing cardiovascular disease ... Cited by 32 - Related articles - Cached - BL Direct - All 8 versions
- ►oxfordjournals.org A Ericsson, B Hamark, TL Powell, T Jansson - Human Reproduction, 2005 - ESHRE BACKGROUND: Placental glucose transport mechanisms in early pregnancy are poorly
understood. The aims of this study were to investigate the expression of glucose
transporter (GLUT) isoforms 1, 3 and 4 in first trimester villous tissue, ... Cited by 26 - Related articles - All 6 versions
- ►physiology.org A Ericsson, B Hamark, N Jansson, BR … - … Journal of Physiology- Regulatory, Integrative and …, 2005 - Am Physiological Soc Alterations in placental nutrient transfer have been implicated in fetal growth
abnormalities. In pregnancies complicated by diabetes and accelerated fetal
growth, upregulations of glucose transporter 1 (GLUT1) and amino acid ... Cited by 23 - Related articles - All 4 versions
- ►endojournals.org M Johansson, L Karlsson, M Wennergren, T … - Journal of Clinical Endocrinology & Metabolism, 2003 - Endocrine Soc In contrast to classical transporting epithelia, the Na + /K + ATPase is
distributed to both the microvillous membrane (MVM) and the basal membrane (BM)
of the placental syncytiotrophoblast. Na + /K + ATPase is important in ... Cited by 22 - Related articles - BL Direct - All 4 versions
M Lampl, P Jeanty - American Journal of Human Biology, 2004 - interscience.wiley.com The prevalence of diabetes is rising worldwide, including women who grew poorly
in early life, presenting intergenerational health problems for their offspring.
It is well documented that fetuses exposed to maternal diabetes during ... Cited by 21 - Related articles - BL Direct - All 2 versions
T Jansson, TL Powell - Placenta, 2006 - Elsevier Intrauterine growth restriction is associated with a range of alterations in
placental transport functions: the activity of a number of transporters is
reduced (Systems A, L and Tau, transporters for cationic amino acids, the ... Cited by 20 - Related articles - All 11 versions
