P Vangheluwe, L Raeymaekers, L Dode, F Wuytack - Cell Calcium, 2005 - Elsevier Of the three mammalian members belonging to the sarco(endo)plasmic reticulum Ca 2+ ATPase
(SERCA) family, SERCA2 is evolutionary the oldest and shows the most wide tissue-expression
pattern. Two major SERCA2 splice variants are well-characterized: the muscle-specific ... Cited by 42 - Related articles - All 4 versions
CS Nunemaker, M Zhang, LS Satin - Diabetes, 2004 - Am Diabetes Assoc Recent work suggests that insulin may exert both positive and negative feedback directly on
pancreatic β-cells. To investigate the hypothesis that insulin modulates β-cell metabolism, mouse
islets and β-cell clusters were loaded with rhodamine 123 to dynamically monitor ... Cited by 15 - Related articles - BL Direct - All 5 versions
- ►endocrinology-journals.org YF Zhao, DJ Keating, M Hernandez, DD … - Journal of molecular …, 2005 - Soc Endocrinology Dysfunction of pancreatic ß-cells is a fundamental feature in the pathogenesis of type 2
diabetes. As insulin receptor signaling occurs via protein tyrosine kinase (PTK), we investigated
the role of PTK activity in the etiology of ß-cell dysfunction by inhibiting PTK activity in ... Cited by 7 - Related articles - BL Direct - All 4 versions
- ►fasebj.org DA Glauser, W Schlegel - The FASEB Journal, 2007 - FASEB The AP-1 transcription factor composed of fos and jun gene products mediates transcriptional
responses to hormonal and metabolic stimulations of pancreatic beta cells. Here, we investigated
the mechanisms that dynamically control expression of AP-1 subunit proteins. In MIN6 ... Cited by 6 - Related articles - BL Direct - All 4 versions
T Korsgaard, M Colding-Jorgensen - Journal of Biological Physics, …, 2006 - ingentaconnect.com Abstract The relation between plasma glucose and insulin release from pancreatic beta- cells
is not stationary in the sense that a given glucose concentration leads to a specific rate of insulin
secretion. A number of time-dependent mechanisms appear to exist that modify insulin ... Cited by 2 - Related articles - BL Direct - All 2 versions
T Vagn Korsgaard, M Colding-Jørgensen - Journal of Biological Physics, 2006 - Springer Abstract The relation between plasma glucose and insulin release from pancreatic beta- cells
is not stationary in the sense that a given glucose concentration leads to a specific rate of insulin
secretion. A number of time-dependent mechanisms appear to exist that modify insulin ... Related articles - All 2 versions
SJ Lee, HE Kim, SE Choi, HC Shin, WJ Kwag, BK Lee, … - Molecules and Cells, 2009 - Springer Genistein has been reported to potentiate glucose- stimulated insulin secretion (GSIS). Inhibitory
activity on tyrosine kinase or activation of protein kinase A (PKA) was shown to play a role in
the genistein-induced poten- tiation effect on GSIS. The aim of the present study was to ... Related articles - All 2 versions
M Düfer, D Haspel, P Krippeit-Drews, L Aguilar- … - Pflügers Archiv European …, 2009 - Springer Abstract Pancreatic beta-cells of sulfonylurea receptor type 1 knock-out (SUR1 −/− ) mice exhibit
an oscillating membrane potential (V m ) demonstrating that hyper-polar- isation occurs despite
the lack of K ATP channels. We hypothesize that glucose activates the Na + /K + -ATPase ... Related articles - All 2 versions
