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Scholar Results 1 - 10 of about 23 citing Campbell: Glucose-induced phosphatidylinositol 3-kinase and mitogen-activated protein kinase-depe.... (0.12 sec) 

Glucose-potentiated chemotaxis in human vascular smooth muscle is dependent on cross- …

- ahajournals.org [PDF] 
M Campbell, WE Allen, C Sawyer, B … - Circulation research, 2004 - Am Heart Assoc
Kluwer Health, 351 West Camden Street, Baltimore, MD 21202-2436. Phone:
410-528-4050. Permissions: Permissions & Rights Desk, Lippincott Williams &
Wilkins, a division of Wolters ... Glucose-Potentiated Chemotaxis in Human ...
Cited by 40 - Related articles - BL Direct - All 6 versions

Modification of PI3K-and MAPK-dependent chemotaxis in aortic vascular smooth muscle cells …

- ahajournals.org [PDF] 
M Campbell, ER Trimble - Circulation research, 2005 - Am Heart Assoc
ISSN: 1524-4571 Copyright © 2005 American Heart Association. All rights
reserved. Print ISSN: 0009-7330. Online TX 72514 Circulation Research is
published by the American Heart Association. 7272 Greenville Avenue, ...
Cited by 36 - Related articles - All 6 versions

Pulmonary arterial hypertension is linked to insulin resistance and reversed by peroxisome …

- ahajournals.org
G Hansmann, RA Wagner, S Schellong, VA de … - Circulation, 2007 - Am Heart Assoc
Methods and Results— We report that apoE –/– mice on a high-fat diet
develop PAH as judged by elevated right ventricular systolic pressure. Compared
with females, male apoE –/– were insulin resistant, had lower plasma ...
Cited by 38 - Related articles - All 12 versions

D-Glucose upregulates adenosine transport in cultured human aortic smooth muscle cells

- physiology.org
GPH Leung, RYK Man, CM Tse - American Journal of Physiology- Heart and Circulatory …, 2005 - Am Physiological Soc
The etiology of the atherosclerosis that occurs in diabetes mellitus is unclear.
Adenosine has been shown to inhibit growth of rat aortic smooth muscle cells.
Nucleoside transporters play an integral role in adenosine function by ...
Cited by 13 - Related articles - All 5 versions

Hyperglycemia alters the responsiveness of smooth muscle cells to insulin-like growth factor- …

- endojournals.org
LA Maile, BE Capps, Y Ling, G Xi, DR … - Endocrinology, 2007 - Endocrine Soc
IGF-I stimulation of smooth muscle cell (SMC) migration and proliferation
requires Vß3 ligand occupancy. We hypothesized that changes in the levels of
extracellular matrix proteins induced by alterations in glucose ...
Cited by 14 - Related articles - BL Direct - All 4 versions

Role of the Integrin αVβ3 in Mediating Increased Smooth Muscle Cell Responsiveness to …


DR Clemmons, LA Maile, Y Ling… - Growth hormone & IGF research: official journal of …, 2007 - pubmedcentral.nih.gov
Under usual conditions, the role of IGF-I in vascular cell types is to maintain
cellular protein synthesis and cell size, and even excess IGF-I does not
stimulate proliferation. In pathophysiologic states, such as hyperglycemia, ...
Cited by 10 - Related articles - All 2 versions

Internal mammary artery smooth muscle cells resist migration and possess high antioxidant …

- oxfordjournals.org
VS Mahadevan, M Campbell, PP McKeown, U … - Cardiovascular research, 2006 - cardiovascres.oxfordjournals.org
Results: Chemotaxis experiments revealed that while both sets of VSMC migrated
towards platelet-derived growth factor-BB (1–50 ng/ml) and angiotensin II
(1–50 nM), neither oxidized-LDL (ox-LDL, 25–100 µg/ml) nor native LDL ...
Cited by 11 - Related articles - All 7 versions

Enhanced cell cycle entry and mitogen-activated protein kinase-signaling and downregulation …


AWY Chung, H Luo, T Tejerina, C van … - Atherosclerosis, 2007 - Elsevier
Diabetic patients have a strong predilection for atherosclerosis and
postangioplasty restenosis. Accelerated cell proliferation and excessive
extracellular matrix deposition are believed to contribute to the ...
Cited by 12 - Related articles - All 31 versions

Glucose regulation of integrin-associated protein cleavage controls the response of vascular …

- endojournals.org
LA Maile, BE Capps, EC Miller, LB Allen, U … - Molecular Endocrinology, 2008 - Endocrine Soc
Vascular smooth muscle cells (SMC) maintained in high glucose are more
responsive to IGF-I than SMC maintained in normal glucose due to a difference in
the Shc phosphorylation response. In this study we aimed to determine the ...
Cited by 7 - Related articles - BL Direct - All 4 versions

Insulin-like growth factor-I stimulates Shc-dependent phosphatidylinositol 3-kinase …


Y Radhakrishnan, LA Maile, Y Ling, LM … - Journal of Biological Chemistry, 2008 - ASBMB
Insulin-like growth factor-I (IGF-I) stimulates vascular smooth muscle cell
proliferation and migration by activating both MAPK and phosphatidylinositol
3-kinase (PI3K). Vascular smooth muscle cells (VSMCs) maintained in 25 mm ...
Cited by 7 - Related articles - All 7 versions


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