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Scholar Results 1 - 10 of about 56 citing Sempoux: The focal form of persistent hyperinsulinemic hypoglycemia of infancy. (0.13 sec) 

Pancreatic endocrine tumors


G Klöppel, M Anlauf - Pathology Case Reviews, 2006 - journals.lww.com
The endocrine tumors of the pancreas are composed of cells with a neuroendocrine
phenotype. Well-differentiated tumors, well-differentiated carcinomas, poorly
differentiated carcinomas, functioning (insulinomas, gastrinomas, ...
Cited by 135 - Related articles - BL Direct - All 4 versions

Hyperinsulinism in infancy: from basic science to clinical disease

- physiology.org
MJ Dunne, KE Cosgrove, RM Shepherd, A … - Physiological reviews, 2004 - Am Physiological Soc
The earliest description of hyperinsulinism appears to have been that provided
by Laidlaw in 1938 who used the term nesidioblastosis to describe the severe,
recurrent hypoglycemia associated with an inappropriate elevation of serum ...
Cited by 108 - Related articles - BL Direct - All 3 versions

Noninvasive diagnosis of focal hyperinsulinism of infancy with [18F]-DOPA positron …

- shouxi.net
T Otonkoski, K Näntö-Salonen, M Seppänen, … - Diabetes, 2006 - Am Diabetes Assoc
Congenital hyperinsulinism of infancy (CHI) is characterized by severe
hypoglycemia due to dysregulated insulin secretion, associated with either focal
or diffuse pathology of the endocrine pancreas. The focal condition is ...
Cited by 77 - Related articles - BL Direct - All 7 versions

KATP channels and insulin secretion disorders

- physiology.org
H Huopio, SL Shyng, T Otonkoski, CG Nichols - American Journal of Physiology- Endocrinology And …, 2002 - Am Physiological Soc
ATP-sensitive potassium (K ATP ) channels are inhibited by intracellular ATP and
activated by ADP. Nutrient oxidation in -cells leads to a rise in [ATP]-to-[ADP]
ratios, which in turn leads to reduced K ATP channel activity, ...
Cited by 61 - Related articles - BL Direct - All 6 versions

Hereditary hormone excess: genes, molecular pathways, and syndromes

- endojournals.org
SJ Marx, WF Simonds - Endocrine reviews, 2005 - Endocrine Soc
Hereditary origin of a tumor helps toward early discovery of its mutated gene;
for example, it supports the compilation of a DNA panel from index cases to
identify that gene by finding mutations in it. The gene for a hereditary ...
Cited by 41 - Related articles - BL Direct - All 5 versions

Persistent Hyperinsulinemic Hypoglycemia in 15 Adults With Diffuse Nesidioblastosis: …


M Anlauf, D Wieben, A Perren, B Sipos, P … - The American journal of surgical pathology, 2005 - journals.lww.com
Abstract: Persistent hyperinsulinemic hypoglycemia (PHH) in adults that is not
caused by an insulinoma is a rare and not well- characterized disease that has
been named nesidioblastosis. In this study, we defined and scrutinized ...
Cited by 41 - Related articles - All 5 versions

Mechanisms of disease: advances in diagnosis and treatment of hyperinsulinism in …


DD De León, CA Stanley - Nature Clinical Practice Endocrinology & Metabolism, 2007 - nature.com
Diva D De León is an Assistant Professor of Pediatrics at the University of
Pennsylvania, Philadelphia, PA, USA, and an Attending Physician in the Division
of Endocrinology, The Children's Hospital of Philadelphia. Her research ...
Cited by 28 - Related articles - BL Direct - All 4 versions

[CITATION] Tumors of the endocrine pancreas


GKPU Heitz - Diagnostic Histopathology of Tumors - Elsevier Health Sciences
Cited by 27 - Related articles - All 4 versions

Molecular mechanisms of neonatal hyperinsulinism


I Giurgea, C Bellanné-Chantelot, M Ribeiro, L … - Horm Res, 2006 - content.karger.com
Congenital hyperinsulinism (CHI), characterized by profound hypoglycaemia
related to inappropriate insulin secretion, may be associated histologically
with either diffuse insulin hypersecretion or focal adenomatous ...
Cited by 21 - Related articles - BL Direct - All 5 versions

Limited therapeutic efficacy of pioglitazone on progression of hepatic fibrosis in rats

- nih.gov
IA Leclercq, C Sempoux, P Starkel, Y … - British Medical Journal, 2006 - gut.bmj.com
Results: When introduced after two weeks of CCl 4 , pioglitazone reduced hepatic
fibrosis, OH proline content, hepatic mRNA expression of collagen type I, and
profibrotic genes, as well as the number of activated smooth muscle actin ...
Cited by 21 - Related articles - BL Direct - All 6 versions


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