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Scholar Results 1 - 10 of about 21 citing El-Haschimi: Insulin resistance and lipodystrophy in mice lacking ribosomal S6 kinase 2. (0.08 sec) 

[PDF] The human obesity gene map: the 2005 update


T Rankinen, A Zuberi, YC Chagnon, SJ … - Obesity, 2006 - pbrc.edu
Abstract RANKINEN, TUOMO, AAMIR ZUBERI, YVON C. CHAGNON, S. JOHN WEISNAGEL,
GEORGE ARGYROPOULOS, BRANDON WALTS, LOUIS PE´RUSSE, AND CLAUDE BOUCHARD. The
human obesity gene map: the 2005 update. Obesity. 2006;14: 529–644. This ...
Cited by 332 - Related articles - View as HTML - All 6 versions

[PDF] The human obesity gene map: the 2004 update


L Perusse, T Rankinen, A Zuberi, YC Chagnon … - Obesity research, 2005 - informatik.hu-berlin.de
Abstract PE´ RUSSE, LOUIS, TUOMO RANKINEN, AAMIR ZUBERI, YVON C. CHAGNON, S.
JOHN WEISNAGEL, GEORGE ARGYROPOULOS, BRANDON WALTS, ERIC E. SNYDER, AND CLAUDE
BOUCHARD. The human obesity gene map: the 2004 update. Obes Res. 2005; ...
Cited by 210 - Related articles - View as HTML - All 6 versions

Intracellular signalling pathways activated by leptin


G Frühbeck - Biochemical Journal, 2006 - pubmedcentral.nih.gov
Leptin is a versatile 16 kDa peptide hormone, with a tertiary structure
resembling that of members of the long-chain helical cytokine family. It is
mainly produced by adipocytes in proportion to fat size stores, and was ...
Cited by 203 - Related articles - BL Direct - All 8 versions

Retinol-binding protein 4 is associated with insulin resistance and body fat distribution in …

- endojournals.org
S Gavi, LM Stuart, P Kelly, MM Melendez, DC … - Journal of Clinical Endocrinology & Metabolism, 2007 - Endocrine Soc
Objectives: The primary objective of this study was to determine whether an
association between retinol-binding protein 4 (RBP4) and insulin resistance
exists in nonobese individuals without a family history or diagnosis of ...
Cited by 57 - Related articles - BL Direct - All 5 versions

Functional characterization of human RSK4, a new 90-kDa ribosomal S6 kinase, reveals …

- shouxi.net
BA Dümmler, C Hauge, J Silber, HG Yntema, … - Journal of Biological Chemistry, 2005 - ASBMB
The 90-kDa ribosomal S6 kinases (RSK1–3) are important mediators of growth
factor stimulation of cellular proliferation, survival, and differentiation and
are activated via coordinated phosphorylation by ERK and 3-phosphoinositide- ...
Cited by 28 - Related articles - All 6 versions

Glucose 6-phosphate causes translocation of phosphorylase in hepatocytes and inactivates …

- nih.gov [PDF] 
S Aiston, A Green, M Mukhtar, L Agius - Biochemical Journal, 2004 - pubmedcentral.nih.gov
The role of glucose 6-P (glucose 6-phosphate) in regulating the activation state
of glycogen synthase and its translocation is well documented. In the present
study, we investigated the effects of glucose 6-P on the activation state ...
Cited by 16 - Related articles - BL Direct - All 8 versions

Deletion of the Coffin–Lowry syndrome gene Rsk2 in mice is associated with impaired …


R Poirier, S Jacquot, C Vaillend, AA … - Behavior genetics, 2007 - Springer
Abstract Coffin–Lowry Syndrome (CLS) is an X-linked syndromic form of mental
retardation associ- ated with skeletal abnormalities. It is caused by muta-
tions of the Rsk2 gene, which encodes a growth factor regulated kinase. ...
Cited by 16 - Related articles - BL Direct - All 4 versions

[PDF] The RSK factors of activating the Ras/MAPK signaling cascade


A Carriere, H Ray, J Blenis, PP Roux - Front Biosci, 2008 - roux.iric.ca
1Institute for Research in Immunology and Cancer (IRIC), and Department of
Pathology and Cell Biology, Faculty of Medicine, Universite de Montreal,
Montreal, Quebec H3C 3J7, Canada, 2Harvard Medical School, Department of ...
Cited by 15 - Related articles - View as HTML - All 2 versions

The p53 protein is a novel substrate of ribosomal S6 kinase 2 and a critical intermediary for …

- aacrjournals.org
YY Cho, Z He, Y Zhang, HS Choi, F Zhu, BY … - Cancer Research, 2005 - AACR
The tumor suppressor p53 protein is one of the most highly connected nodes in
cellular signal transduction pathways and acts as a central regulatory switch in
networks controlling cell proliferation and apoptosis. It is involved in ...
Cited by 14 - Related articles - All 3 versions

Mouse models of lipodystrophy: key reagents for the understanding of the metabolic …

- nih.gov
IW Asterholm, N Halberg, PE Scherer - Drug Discovery Today: Disease Models, 2007 - Elsevier
Both the disproportionate loss of adipose tissue in the case of lipodystrophies
and the disproportionate gain of adipose tissue in obesity are frequently
associated with an increase in insulin resistance and its complications. ...
Cited by 8 - Related articles - All 3 versions


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