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Scholar Results 1 - 10 of about 50 citing Santana: A role for advanced glycation end products in diminished bone healing in type 1 diabetes. (0.08 sec) 

Advanced glycation endproducts—role in pathology of diabetic complications


N Ahmed - Diabetes research and clinical practice, 2005 - Elsevier
Diabetes mellitus is a common endocrine disorder characterised by hyperglycaemia
and predisposes to chronic complications affecting the eyes, blood vessels,
nerves and kidneys. Hyperglycaemia has an important role in the ...
Cited by 209 - Related articles - All 10 versions

Osteoporosis and cardiovascular disease


SI McFarlane, R Muniyappa, JJ Shin, G … - Endocrine, 2004 - Springer
Received December 22, 2003; Revised January 28, 2004; Accepted January 30, 2004.
Author to whom all correspondence and reprint requests should be addressed: Samy
I. McFarlane, MD, FACP, Associate Professor of Medicine, Fellow- ship ...
Cited by 79 - Related articles - BL Direct - All 3 versions

Is insulin an anabolic agent in bone? Dissecting the diabetic bone for clues

- physiology.org
KM Thrailkill, CK Lumpkin Jr, RC Bunn, SF … - American Journal of Physiology- Endocrinology And …, 2005 - Am Physiological Soc
Diabetic osteoporosis is increasingly recognized as a significant comorbidity of
type 1 diabetes mellitus. In contrast, type 2 diabetes mellitus is more commonly
associated with modest increases in bone mineral density for age. Despite ...
Cited by 54 - Related articles - BL Direct - All 5 versions

Advanced glycation end products enhance expression of pro-apoptotic genes and stimulate …

- shouxi.net
Z Alikhani, M Alikhani, CM Boyd, K Nagao, … - Journal of Biological Chemistry, 2005 - ASBMB
Both aging and diabetes are characterized by the formation of advanced glycation
end products (AGEs). Both exhibit other similarities including deficits in wound
healing that are associated with higher rates of fibroblast apoptosis. In ...
Cited by 47 - Related articles - All 6 versions

Diabetes-enhanced inflammation and apoptosis--impact on periodontal pathology

- sagepub.com
DT Graves, R Liu, M Alikhani, H Al-Mashat, … - Journal of dental research, 2006 - jdr.sagepub.com
Cited by 31 - Related articles - BL Direct - All 3 versions

HMGB1 expression and release by bone cells


K Charoonpatrapong, R Shah, AG Robling, M … - Journal of cellular physiology, 2006 - interscience.wiley.com
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Cited by 30 - Related articles - BL Direct - All 5 versions

Advanced glycation end products stimulate osteoblast apoptosis via the MAP kinase and …

- nih.gov
M Alikhani, Z Alikhani, C Boyd, CM MacLellan … - Bone, 2007 - Elsevier
We have previously shown that diabetes significantly enhances apoptosis of
osteoblastic cells in vivo and that the enhanced apoptosis contributes to
diabetes impaired new bone formation. A potential mechanism is enhanced ...
Cited by 29 - Related articles - All 16 versions

Disordered osteoclast formation in RAGE-deficient mouse establishes an essential role for …

- 211.64.32.2 [PDF] 
KH Ding, ZZ Wang, MW Hamrick, ZB Deng, L … - Biochemical and Biophysical Research …, 2006 - Elsevier
The mechanisms underlying diabetes-mediated bone loss are not well defined. It
has been reported that the advanced glycation endproducts (AGEs) and receptor
for AGEs (RAGEs) are involved in diabetic complications. Here, mice ...
Cited by 21 - Related articles - All 7 versions

Bone loss and increased bone adiposity in spontaneous and pharmacologically induced …

- endojournals.org
S Botolin, LR McCabe - Endocrinology, 2007 - Endocrine Soc
Insulin-dependent diabetes mellitus (IDDM) is associated with increased risk of
osteopenia/osteoporosis in humans. The mechanisms accounting for diabetic bone
loss remain unclear. Pharmacologic inducers of IDDM, such as ...
Cited by 21 - Related articles - BL Direct - All 5 versions

Influence of diabetic metabolic state on fracture healing in spontaneously diabetic rats


N Follak, I Kloting, H Merk - Diabetes/Metabolism Reviews - interscience.wiley.com
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Cited by 21 - Related articles - All 3 versions


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