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Scholar Results 1 - 10 of about 37 citing Anderson: Production of Nε-(Carboxymethyl) Lysine Is Impaired in Mice Deficient in NADPH Oxidase. (0.07 sec) 

Advanced glycoxidation end products in chronic diseases—clinical chemistry and genetic …


M Kalousova, T Zima, V Tesař, S Dusilová- … - … Research-Fundamental and Molecular Mechanisms of …, 2005 - Elsevier
Several diseases (atherosclerosis, diabetes mellitus, chronic renal failure) are
associated with oxidative and carbonyl stress, microinflammation and eventually
autoimmune reaction. Both oxidative and carbonyl stress cause damage to ...
Cited by 51 - Related articles - All 5 versions

[PDF] Immunity and aging: the enemy within?


B DeVeale, T Brummel, L Seroude… - Aging Cell, 2004 - seroudelab.biology.queensu.ca
The middle-age crisis GAL4 enhancer-trap is inserted in between two genes
encoding peptidoglycan recognition proteins (PGR-LC and PGRP-LF). GAL4
expression is controlled by the same age-dependent regulatory sequences ...
Cited by 48 - Related articles - View as HTML - BL Direct - All 4 versions

Diabetes and advanced glycoxidation end products

- diabetesjournals.org
… JG Regensteiner, H Vlassara, JEB Reusch - Diabetes Care, 2006 - Am Diabetes Assoc
The morbidity caused by diabetes has traditionally been classified into macro-
and microvascular complications. Although macrovascular complications have
received greater attention, microvascular complications are unique to ...
Cited by 41 - Related articles - All 6 versions

Vascular and inflammatory stresses mediate atherosclerosis via RAGE and its ligands in apoE …

- nih.gov
E Harja, D Bu, BI Hudson, JS Chang, X Shen, … - Journal of Clinical Investigation, 2007 - Am Soc Clin Investig
Endothelial dysfunction is a key triggering event in atherosclerosis. Following
the entry of lipoproteins into the vessel wall, their rapid modification results
in the generation of advanced glycation endproduct epitopes and subsequent ...
Cited by 38 - Related articles - BL Direct - All 5 versions

Receptor for advanced glycation end products (RAGE) in a dash to the rescue: inflammatory …

- jleukbio.org
K Herold, B Moser, Y Chen, S Zeng, SF Yan, … - Journal of leukocyte biology, 2007 - Soc Leukocyte Biology
The multiligand receptor for advanced glycation end products (RAGE) of the Ig
superfamily transduces the biological impact of discrete families of ligands,
including advanced glycation end products, certain members of the S100/ ...
Cited by 31 - Related articles - BL Direct - All 7 versions

Measurement of N {epsilon}-(carboxymethyl) lysine and N {epsilon}-(carboxyethyl) lysine …

- clinchem.org
T Teerlink, R Barto, HJ ten Brink, CG … - Clinical Chemistry, 2004 - Am Assoc Clin Chem
Methods: CML and CEL were liberated from plasma proteins by acid hydrolysis
after addition of deuterated CML and CEL as internal standards. Chromatographic
separation was performed by gradient-elution reversed-phase chromatography ...
Cited by 27 - Related articles - All 5 versions

Advanced glycation end products and nephrotoxicity of high-protein diets

- asnjournals.org
J Uribarri, KR Tuttle - Clinical Journal of the American Society of Nephrology, 2006 - Am Soc Nephrol
The popularity of high-protein diets has surged recently as obesity has become
more and more common in the United States and other developed nations. In view
of the high prevalence of type 2 diabetes and chronic kidney disease among ...
Cited by 17 - Related articles - BL Direct - All 3 versions

Receptor for Advanced Glycation Endproducts (RAGE): A Formidable Force in the …


R Ramasamy, SF Yan, V DAgati, AM Schmidt - Current Molecular Medicine, 2007 - ingentaconnect.com
Departments of Surgery 1 and Pathology 2 , Columbia University Medical Center,
New York, NY 10032, USA Abstract: Unifying mechanisms for the consequences of
aging and chronic diabetes are coming to light with the identification that ...
Cited by 17 - Related articles - BL Direct - All 2 versions

Role of glucoxidation and lipid oxidation in the development of atherosclerosis


E Schleicher, C Weigert, H Rohrbach, A … - ANNALS-NEW YORK ACADEMY OF SCIENCES, 2005 - interscience.wiley.com
Abstract: Previous data have indicated that modification of proteins/lipids by
glucoxidation and/or lipid oxidation may initiate/propagate the formation of
atherosclerotic plaques. Although the biomarker carboxymethyllysine (CML) ...
Cited by 16 - Related articles - BL Direct - All 5 versions

Protein damage and inflammation in uraemia and dialysis patients

- oxfordjournals.org
F Galli - Nephrology Dialysis Transplantation, 2007 - ERA-EDTA
The presence of high molecular weight toxins in the uraemic blood had largely
been ignored until the beginning of the era of proteomics. In the last decade,
increasing interest focused on these solutes has yielded evidence to ...
Cited by 14 - Related articles - BL Direct - All 7 versions


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