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Scholar Results 1 - 10 of about 46 citing Catrina: Hyperglycemia regulates hypoxia-inducible factor-1α protein stability and function. (0.09 sec) 

Homing to hypoxia: HIF-1 as a mediator of progenitor cell recruitment to injured tissue

- spinesmithusa.com [PDF] 
DJ Ceradini, GC Gurtner - Trends in Cardiovascular Medicine, 2005 - Elsevier
The identification of bone marrow-derived endothelial progenitor cells has
altered our understanding of new blood vessel growth and tissue regeneration.
Previously, new blood vessel growth in the adult was thought to only occur ...
Cited by 106 - Related articles - All 13 versions

Diabetes impairs progenitor cell mobilisation after hindlimb ischaemia–reperfusion injury in …


GP Fadini, S Sartore, M Schiavon, M Albiero, … - Diabetologia, 2006 - Springer
Page 1. ARTICLE Diabetes impairs progenitor cell mobilisation after hindlimb
ischaemia–reperfusion injury in rats GP Fadini & S ...
Cited by 43 - Related articles - BL Direct - All 5 versions

Anti-tumor necrosis factor therapy increases synovial osteoprotegerin expression in …


AI Catrina, E af Klint, S Ernestam, SB … - Arthritis Care & Research - interscience.wiley.com
It is also possible that your web browser is not configured or not able to
display style sheets. In this case, although the visual presentation will be
degraded, the site should continue to be functional. We recommend using the ...
Cited by 38 - Related articles - BL Direct - All 4 versions

High glucose blunts vascular endothelial growth factor response to hypoxia via the …

- asnjournals.org
P Katavetin, T Miyata, R Inagi, T Tanaka, R … - Journal of the American Society of Nephrology, 2006 - Am Soc Nephrol
Vascular endothelial growth factor (VEGF) is an important survival factor for
endothelial cells in hypoxic environments. High glucose regulates certain
aspects of VEGF expression in various cell types, including proximal ...
Cited by 28 - Related articles - BL Direct - All 5 versions

Regulation of hypoxia-inducible factor-1α by NF-κB


P van Uden, NS Kenneth, S Rocha - Biochemical Journal, 2008 - pubmedcentral.nih.gov
This is an open-access article distributed under the terms of the Creative
Commons Attribution License, which permits unrestricted use, distribution, and
reproduction in any medium, provided the original work is properly cited.
Cited by 23 - Related articles - BL Direct - All 6 versions

Embryonic oxidative stress as a mechanism of teratogenesis with special emphasis on …


A Ornoy - Reproductive Toxicology, 2007 - Elsevier
Reactive oxygen species (ROS) are involved in the etiology of numerous diseases
including cardio-vascular diseases and diabetes mellitus. There is evidence that
several teratogens affect the developing embryo by increasing its oxidative ...
Cited by 21 - Related articles - All 6 versions

Chronic hyperglycemia modulates osteoblast gene expression through osmotic and non- …


S Botolin, LR McCabe - Journal of cellular biochemistry, 2006 - interscience.wiley.com
Insulin dependent diabetes mellitus (IDDM; type I) is a chronic disease stemming
from little or no insulin production and elevated blood glucose levels. IDDM is
associated with osteoporosis and increased fracture rates. The mechanisms ...
Cited by 21 - Related articles - BL Direct - All 4 versions

Endogenous hypoxia markers in locally advanced cancers of the uterine cervix: reality or …


A Mayer, M Höckel, P Vaupel - Strahlentherapie und Onkologie, 2006 - Springer
Arnulf Mayer 1 ,Michael Höckel 2 ,Peter Vaupel 1 ... Background: Tumor hypoxia
is considered to be relevant for several aspects of tumor pathophysiology, for
acquired treatment resistance, and tumor progression (eg, in cancers of the ...
Cited by 20 - Related articles - BL Direct - All 3 versions

Hypoxia-inducible factor-1α and hypoxia-inducible factor-2α are expressed in Kaposi …


SB Catrina, IR Botusan, A Rantanen, AI … - Clinical Cancer Research, 2006 - AACR
Purpose: Neoangiogenesis is essential for tumor development. Hypoxia-inducible
factor (HIF), a transcriptional factor composed of two subunits (α and β),
plays a key role in this process, activating proangiogenic factors such as ...
Cited by 17 - Related articles - BL Direct - All 4 versions

Protein kinase C {beta} inhibition: a novel therapeutic strategy for diabetic microangiopathy

- dvdres.com [PDF] 
I Idris, R Donnelly - Diabetes & Vascular Disease Research, 2006 - dvr.sagepub.com
Abstract B iochemical mechanisms involved in hyperglycaemia- induced vascular
damage include alterations in cellular signalling by activation of protein
kinase C (PKC). Twelve isoforms of PKC have been characterised according to ...
Cited by 16 - Related articles - BL Direct - All 8 versions


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