- ►physiology.org KE Knoll, JL Pietrusz, M Liang - Physiological Genomics, 2005 - Am Physiological Soc The understanding of common and tissue-specific molecular alterations in
diabetes, particularly at early stages, is limited and fragmental. In the
present study, we systematically compared transcriptome responses in four ... Cited by 29 - Related articles - All 5 versions
- ►mcponline.org T Sparre, MR Larsen, PE Heding, AE Karlsen, … - Molecular & Cellular Proteomics, 2005 - ASBMB Type 1 diabetes (T1D) is the result of selective destruction of the
insulin-producing ß-cells in the pancreatic islets of Langerhans. T1D is due to
a complex interplay between the ß-cell, the immune system, and the ... Cited by 21 - Related articles - BL Direct - All 6 versions
- ►physiology.org [PDF] SS Vukkadapu, JM Belli, K Ishii, AG Jegga, JJ … - Physiological Genomics, 2005 - Am Physiological Soc Page 1. Dynamic interaction between T cell-mediated beta cell damage and beta cell
repair in the run-up to autoimmune diabetes of the NOD mouse ... Cited by 21 - Related articles - All 4 versions
SY Killedar, SE Eckenrode, RA McIndoe, JX … - Laboratory Investigation, 2006 - nature.com Recently, we reported development of the C57BL/6.NOD-Aec1Aec2 mouse carrying two
genetic intervals derived from the NOD mouse. These two genetic regions confer
full Sjögren's syndrome (SjS)-like disease in SjS-non-susceptible C57BL/6 ... Cited by 13 - Related articles - BL Direct - All 5 versions
- ►diabetesjournals.org UC Rogner, F Lepault, MC Gagnerault, D … - Diabetes, 2006 - Am Diabetes Assoc The genetic locus Idd6 confers susceptibility to the spontaneous development of
type 1 diabetes in the NOD mouse. Our studies on disease resistance of the
congenic mouse strain NOD.C3H 6.VIII showed that Idd6 influences T-cell ... Cited by 10 - Related articles - BL Direct - All 7 versions
- ►oxfordjournals.org MS Hung, P Avner, UC Rogner - Human Molecular Genetics, 2006 - Oxford Univ Press The Idd6 murine type 1 diabetes locus has been shown to control diabetes by
regulating the protective activity of the peripheral immune system, as
demonstrated by diabetes transfer assays using splenocytes. The analysis of ... Cited by 8 - Related articles - BL Direct - All 6 versions
- ►shouxi.net Y Kagohashi, J Udagawa, N Abiru, M … - Diabetes, 2005 - Am Diabetes Assoc Type 1 diabetes, a multifactorial disease involving genetic and environmental
factors, results from the destruction of pancreatic β-cells. The maternal
environment has been suggested to be important in the development of ... Cited by 8 - Related articles - All 5 versions
- ►googlepages.com [PDF] M Balasubramanyam, R Sampathkumar, V … - Molecular and cellular biochemistry, 2005 - Springer Recent mining of the human and mouse genomes, use of yeast genetics, and
detailed analyses of several biochemical pathways, have resulted in the
identification of many new roles for ubiquitin–proteasome mediated ... Cited by 7 - Related articles - BL Direct - All 7 versions
J Morin, C Boitard, D Vallois, P Avner, UC … - Mammalian Genome, 2006 - Springer Abstract In the nonobese diabetes mouse, the murine type 1 diabetes
susceptibility locus Idd20 interacts geneti- cally with the diabetes resistance
locus Idd19. Both Idds are located on distal mouse Chromosome 6, and ... Cited by 4 - Related articles - BL Direct - All 4 versions
SE Eckenrode, QG Ruan, CD Collins, P Yang, … - Ann NY Acad Sci, 2004 - cmbi.bjmu.edu.cn ABSTRACT: Type 1 diabetes (T1D) in the nonobese diabetic (NOD) mouse can be
delayed by administration of insulin or specific insulin peptides. To better
understand how insulin treatment delays diabetes development, NOD mice ... Cited by 3 - Related articles - View as HTML - All 9 versions
