- ►asnjournals.org MD Breyer, E Bottinger, FC Brosius III, TM … - Journal of the American Society of Nephrology, 2005 - Am Soc Nephrol Mice provide an experimental model of unparalleled flexibility for studying
mammalian diseases. Inbred strains of mice exhibit substantial differences in
their susceptibility to the renal complications of diabetes. Much remains ... Cited by 104 - Related articles - All 6 versions
SD Ricardo, JA Deane - Nephrology, 2005 - med.monash.edu Correspondence: Dr Sharon Ricardo, Department of Anatomy and Cell Biology and
Monash Immunology and Stem Cell Laboratories (MISCL), Monash University,
Clayton, Victoria 3800, Australia. Email: Sharon.ricardo@med.monash.edu.au ... Cited by 27 - Related articles - View as HTML - All 4 versions
MS Simonson - Kidney International, 2007 - nature.com The cause of renal fibrosis in diabetic nephropathy is widely believed to be
phenotypic switching of fibroblasts to an activated state. However, emerging
evidence suggests that diabetes also alters the phenotype of normal, non- ... Cited by 26 - Related articles - BL Direct - All 3 versions
F Zheng, YJ Zeng, AR Plati, SJ Elliot, M Berho, … - Kidney international, 2006 - nature.com The accumulation of advanced glycation end products (AGE) is a key factor in
diabetic nephropathy (DN). Pyridoxamine inhibits AGE formation and protects
against type I DN. Herein we tested: (1) whether C57BL6 db/db mice as a ... Cited by 15 - Related articles - BL Direct - All 5 versions
TQ Nguyen, H Chon, FA van Nieuwenhoven, … - Diabetologia, 2006 - Springer Abstract Aims/hypothesis: Growth factor imbalance and endothelial progenitor
cell dysfunction are well-known elements of the inappropriate response to injury
in human and experimental diabetes. We hypothesised that in diabetes the ... Cited by 13 - Related articles - BL Direct - All 3 versions
- ►amjpathol.org Z Feng, AR Plati, Q Cheng, M Berho, A Banerjee … - American Journal of Pathology, 2005 - ASIP The glomeruli of postmenopausal C57BL6 mice, and age-matched males, show
progressive hypertrophy and glomerulosclerosis. We asked whether this was a
multistage process, was due to alterations in glomerular progenitors, and ... Cited by 11 - Related articles - BL Direct - All 6 versions
- ►diabetesjournals.org ML Brezniceanu, F Liu, CC Wei, I Chénier, N … - Diabetes, 2008 - Am Diabetes Assoc RESULTS—db/db mice developed obesity, hyperglycemia, hypertension, and
albuminuria. In contrast, db/db rCAT-Tg mice became obese and hyperglycemic but
had normal blood pressure and attenuated albuminuria compared with db/db ... Cited by 12 - Related articles - BL Direct - All 3 versions
C Vigneau, F Zheng, K Polgar, PD Wilson, G … - Current opinion in nephrology and hypertension, 2006 - journals.lww.com Recent findings: New studies on embryonic stem cells show that they can be use
to enrich for specific renal progenitors, which integrate into mature
structures. Studies on adult stem cells show that almost all kidney cell ... Cited by 11 - Related articles - BL Direct - All 5 versions
FC Brosius - Reviews in endocrine & metabolic disorders, 2008 - Springer Abstract Progression of diabetic nephropathy (DN) is manifested by gradual
scarring of both the renal glomerulus and tubulointerstitial region. Over the
past several years, the general understanding of the pathogenic factors ... Cited by 6 - Related articles - All 3 versions
