J Nicolay, J Schneider, O Niemoeller, F Artunc … - Cell Physiol Biochem, 2006 - content.karger.com Page 1. 223 Original Paper Cell Physiol Biochem 2006;18:223-232 Accepted:
July 27, 2006 Cellular Physiology and Biochemistry Copyright ... Cited by 38 - Related articles - BL Direct - All 4 versions
- ►diabetesjournals.org D Dobler, N Ahmed, L Song, KE Eboigbodin, … - Diabetes, 2006 - Am Diabetes Assoc Chronic vascular disease in diabetes is associated with disruption of
extracellular matrix (ECM) interactions with adherent endothelial cells,
compromising cell survival and impairing vasculature structure. Loss of ... Cited by 31 - Related articles - BL Direct - All 5 versions
- ►jbc.org SV Dandapani, H Sugimoto, BD Matthews, RJ … - Journal of Biological Chemistry, 2007 - ASBMB Mutations in the -actinin-4 gene ACTN4 cause an autosomal dominant human kidney
disease. Mice deficient in -actinin-4 develop a recessive phenotype
characterized by kidney failure, proteinuria, glomerulosclerosis, and ... Cited by 30 - Related articles - All 5 versions
PY Chuang, Q Yu, W Fang, J Uribarri, JC He - Kidney International, 2007 - nature.com Advanced glycation endproducts (AGEs) and a receptor for AGEs (RAGE) have been
linked in the pathogenesis of diabetic nephropathy. RAGE is usually localized to
podocytes and is increased in diabetes. RAGE activation increases reactive ... Cited by 18 - Related articles - BL Direct - All 3 versions
PJ Thornalley - Trends in Molecular Medicine, 2006 - Elsevier Overexpression and silencing of glyoxalase 1 (Glo1) in brains of mice and
behavioural analyses have suggested a link between Glo1 and anxiety, making Glo1
a possible novel target for anxiolytic-drug development. However, this ... Cited by 17 - Related articles - All 7 versions
- ►googlepages.com [PDF] … , M Balasubramanyam, M Rema, V Mohan - Diabetic Medicine, 2006 - pt.wkhealth.com Aims: While the downstream effects of increased reactive oxygen species (ROS) in
the pathogenesis of diabetes were well studied, only a few studies have explored
the cellular sources of ROS. We examined whether protection against ... Cited by 15 - Related articles - BL Direct - All 7 versions
T Chang, L Wu - Canadian journal of physiology and pharmacology, 2006 - article.pubs.nrc-cnrc.gc.ca Abstract: Pathogenic mechanisms for essential hypertension are unclear despite
striking efforts from numerous research teams over several decades. Increased
production of reactive oxygen species (ROS) has been associated with the ... Cited by 15 - Related articles - BL Direct - All 7 versions
- ►adisonline.com [PDF] ME Williams - Drugs, 2006 - ingentaconnect.com Despite the worldwide epidemic of chronic kidney disease complicating Abstract
diabetes mellitus, current therapies directed against nephroprogression are
limited to angiotensin conversion or receptor blockade. Nonetheless, ... Cited by 16 - Related articles - BL Direct - All 8 versions
- ►physiology.org [PDF] H Xu, L Zeng, H Peng, S Chen, J Jones, TL … - American Journal of Physiology- Renal Physiology, 2006 - Am Physiological Soc 1Division of Nephrology/Hypertension, 2Dept. of Cell and Molecular Biology,
Feinberg School of Medicine, Chicago, IL, 3Division of Cardiovascular Medicine,
Yale School of Medicine, 4Dept. of Pathology, Northwestern University, ... Cited by 14 - Related articles - All 5 versions
R Zent, X Yan, Y Su, BG Hudson, DB Borza, … - Kidney international, 2006 - nature.com Excessive glomerular collagen IV and reactive oxygen species (ROS) production
are key factors in the development of diabetic nephropathy. Integrin 1 1, the
major collagen IV receptor, dowregulates collagen IV and ROS production, ... Cited by 14 - Related articles - BL Direct - All 3 versions
