- Free from Publisher R Meerwaldt, HL Lutgers, TP Links, R Graaff … - Diabetes Care, 2007 - Am Diabetes Assoc OBJECTIVE—Advanced glycation end products (AGEs) are biomarkers of metabolic
stress and are thought to contribute to the increase of coronary heart disease
(CHD) in diabetes. Tissue autofluorescence is related to the accumulation ... Cited by 33 - Related articles - BL Direct - All 6 versions
A Girach, D Manner, M Porta - International Journal of Clinical Practice, 2006 - medscape.com People with diabetes have an increased risk of developing microvascular
complications, diabetic retinopathy, diabetic nephropathy and diabetic
neuropathy, which, if undetected or left untreated, can have a devastating ... Cited by 29 - Related articles - BL Direct - All 8 versions
MA Ihnat, JE Thorpe, A Ceriello - Diabetic Medicine, 2007 - ccmjournal.org Large randomized studies have established that early intensive glycaemic control
reduces the risk of diabetic complications, both micro- and macrovascular.
However, epidemiological and prospective data support a long-term influence ... Cited by 26 - Related articles - BL Direct - All 5 versions
ME Williams, WK Bolton, RG Khalifah, TP … - Am J Nephrol, 2007 - content.karger.com Background/Aims: Treatments of diabetic nephropathy (DN) delay the onset of
end-stage renal disease. We report the results of safety/tolerability studies in
patients with overt nephropathy and type 1/type 2 diabetes treated with ... Cited by 22 - Related articles - BL Direct - All 5 versions
M Busch, S Franke, G Wolf, A Brandstädt, U … - American Journal of Kidney Diseases, 2006 - Elsevier Background: Advanced glycation end products (AGEs) are implicated in the
pathogenesis of vascular damage, especially in patients with diabetes and renal
insufficiency. The oxidatively formed AGE N ε -carboxymethyllysine (CML) ... Cited by 19 - Related articles - All 3 versions
MR Nicolls, K Haskins, SC Flores - Antioxidants & Redox Signaling, 2007 - liebertonline.com T HE VASCULAR endothelium, although not traditionally considered an organ, is
intimately involved in maintain- ing organ and tissue homeostasis, and is a
dynamic structure that responds to local changes in the environment. It ... Cited by 16 - Related articles - BL Direct - All 4 versions
K Fukami, S Yamagishi, S Ueda, S Okuda - Current pharmaceutical design, 2008 - ingentaconnect.com Abstract: Diabetic nephropathy is the most common cause of end-stage renal
disease in the world, and accounts for a significant increase in morbidity and
mortality in patients with diabetes. Therapeutic options such as strict ... Cited by 15 - Related articles - BL Direct - All 3 versions
BK Kilhovd, A Juutilainen, S Lehto, T … - Diabetologia, 2007 - Springer Abstract Aims/hypothesis AGEs, modification products formed by glycation or
glycoxidation of proteins and lipids, have been linked to premature
atherosclerosis in patients with diabetes. We investigated whether ... Cited by 13 - Related articles - BL Direct - All 4 versions
- ►fasebj.org JV Glenn, JR Beattie, L Barrett, N Frizzell, … - The FASEB Journal, 2007 - FASEB The modification of proteins by nonenzymatic glycation leading to accumulation
of advanced glycation end products (AGEs) is a well-established phenomenon of
aging. In the eyes of elderly patients, these adducts have been observed in ... Cited by 9 - Related articles - BL Direct - All 5 versions
- ►physiology.org W Cai, JC He, L Zhu, X Chen, GE Striker, H … - American Journal of Physiology- Cell Physiology, 2008 - Am Physiological Soc Advanced glycation end products (AGEs) promote reactive oxygen species (ROS)
formation and oxidant stress (OS) in diabetes and aging-related diseases.
AGE-induced OS is suppressed by AGER1, an AGE-receptor that counteracts ... Cited by 9 - Related articles - BL Direct - All 2 versions
