- ►jimmunol.org DV Serreze, CM Choisy-Rossi, AE Grier, TM … - The Journal of Immunology, 2008 - Am Assoc Immnol When expressed in NOD, but not C57BL/6 (B6) genetic background mice, the common
class I variants encoded by the H2 g7 MHC haplotype aberrantly lose the ability
to mediate the thymic deletion of autoreactive CD8 + T cells contributing ... Cited by 3 - Related articles - All 2 versions
- ►jimmunol.org D Vallois, MC Gagnerault, P Avner, UC Rogner … - The Journal of Immunology, 2008 - Am Assoc Immnol The number and function of immunoregulatory invariant NKT (iNKT) cells are
genetically controlled. A defect of iNKT cell ontogeny and function has been
implicated as one causal factor of NOD mouse susceptibility to type 1 ... Related articles - All 2 versions
- ►jimmunol.org A Ueno, J Wang, L Cheng, JS Im, Y Shi, SA … - The Journal of Immunology, 2008 - Am Assoc Immnol Semi-invariant NK T cell (iNKT) deficiency has long been associated with the
pathogenesis of type 1 diabetes (T1D), but the linkage between this the
deficiency and T1D susceptibility gene(s) remains unclear. We analyzed NOD ... Related articles - All 2 versions
M Lundholm - umu.diva-portal.org Type 1 Diabetes (T1D) is an autoimmune disorder caused by both genetic and
environmental factors. The non-obese diabetic (NOD) mouse is one of the best and
most commonly studied animal models for T1D. This mouse strain ... Related articles - View as HTML