- ►ahajournals.org G Seebohm, N Strutz-Seebohm, R Birkin, G Dell, C … - Circulation …, 2007 - Am Heart Assoc From the Department of Physiology I (GS, NS-S., RB, AFM, GK, FL), University of Tuebingen,
Germany; Department of Biochemistry (RB, GD, JMT), School of Medical Sciences, University
of Bristol, England; Dipartimento di Scienze e Tecnologie Biologiche ed Ambientali (CB, ... Cited by 35 - Related articles - All 5 versions
- ►diabetesjournals.org F Ranta, D Avram, S Berchtold, M Düfer, G Drews, F … - Diabetes, 2006 - Am Diabetes Assoc Glucocorticoid excess induces hyperglycemia, which may result in diabetes. The present experiments
explored whether glucocorticoids trigger apoptosis in insulin-secreting cells. Treatment of mouse
β-cells or INS-1 cells with the glucocorticoid dexamethasone (0.1 μmol/l) over 4 days in ... Cited by 31 - Related articles - BL Direct - All 6 versions
M Tessier, JR Woodgett - Journal of cellular biochemistry, 2006 - interscience.wiley.com The phosphatidylinositol 3 kinase (PI3K)-signaling pathway plays a critical role in a variety of
cellular responses such as modulation of cell survival, glucose homeostasis, cell division, and
cell growth. PI3K generates important lipid second messengers - phosphatidylinositides ... Cited by 27 - Related articles - BL Direct - All 4 versions
- ►physiology.org F Lang, C Bohmer, M Palmada, G Seebohm, … - Physiological …, 2006 - Am Physiological Soc The serum- and glucocorticoid-inducible kinase-1 (SGK1) is ubiquitously expressed and under
genomic control by cell stress (including cell shrinkage) and hormones (including gluco- and
mineralocorticoids). Similar to its isoforms SGK2 and SGK3, SGK1 is activated by insulin ... Cited by 20 - Related articles - BL Direct - All 4 versions
- ►endojournals.org H Oberkofler, K Klein, TK Felder, F Krempler, W Patsch - Endocrinology, 2006 - Endocrine Soc A role of uncoupling protein 2 (UCP2) as negative modulator of insulin secretion has been
suggested, but the transcriptional pathways regulating ß-cell UCP2 gene expression have been
established in rodents only. We show here that the underlying sequence motifs are not ... Cited by 16 - Related articles - BL Direct - All 3 versions
KM Boini, AM Hennige, DY Huang, B Friedrich, M … - Diabetes, 2006 - Am Diabetes Assoc Excess salt intake decreases peripheral glucose uptake, thus impairing glucose tolerance. Stimulation
of cellular glucose uptake involves phosphatidylinositide-3-kinase (PI-3K)–dependent activation
of protein kinase B/Akt. A further kinase downstream of PI-3K is serum- and glucocorticoid- ... Cited by 13 - Related articles - BL Direct - All 6 versions
S Ullrich, Y Zhang, D Avram, F Ranta, D Kuhl, HU … - … and Biophysical Research …, 2007 - Elsevier Glucocorticoids blunt insulin release, an effect partially due to activation of Kv channels. Similar
to those channels Na + /K + ATPase activity repolarizes the plasma membrane. The present
study explored whether glucocorticoids increase the Na + /K + ATPase activity in ... Cited by 10 - Related articles - All 5 versions
R Rexhepaj, F Artunc, F Grahammer, O Nasir, C … - Pflügers Archiv European …, 2006 - Springer Page 1. ION CHANNELS SGK1 is not required for regulation of colonic ENaC activity
Rexhep Rexhepaj & Ferruh Artunc & Florian Grahammer & Omaima Nasir & Ciprian
Sandu & Björn Friedrich & Dietmar Kuhl & Florian Lang ... Cited by 8 - Related articles - BL Direct - All 3 versions
WS Zawalich, GJ Tesz, H Yamazaki, KC Zawalich, W … - Metabolism, 2006 - Elsevier Dexamethasone inhibits insulin secretion from isolated islets. In the present experiments, possible
underlying biochemical mechanisms responsible for defective secretion were explored. Dexamethasone
(1 μmol/L) had no immediate deleterious effect on 15 mmol/L glucose-induced insulin ... Cited by 7 - Related articles - All 17 versions
