- ►shouxi.net R Masia, JC Koster, S Tumini, F Chiarelli, C … - Diabetes, 2007 - Am Diabetes Assoc Mutations in the pancreatic ATP-sensitive K + channel (K ATP channel) cause
permanent neonatal diabetes mellitus (PNDM) in humans. All of the K ATP channel
mutations examined result in decreased ATP inhibition, which in turn is ... Cited by 17 - Related articles - BL Direct - All 6 versions
- ►physiology.org FM Ashcroft - American Journal of Physiology- Endocrinology And …, 2007 - Am Physiological Soc This essay is based on a lecture given to the American Physiological Society in
honor of Walter B. Cannon, an advocate of homeostasis. It focuses on the role of
the ATP-sensitive potassium K + (K ATP ) channel in glucose homeostasis ... Cited by 12 - Related articles - BL Direct - All 2 versions
CA Girard, FT Wunderlich, K Shimomura, S … - The Journal of Clinical Investigation, 2009 - pubmedcentral.nih.gov Neonatal diabetes is a rare monogenic form of diabetes that usually presents
within the first six months of life. It is commonly caused by gain-of-function
mutations in the genes encoding the Kir6.2 and SUR1 subunits of the ... Cited by 3 - Related articles - All 10 versions
- ►adisonline.com [PDF] BF Palmer - American Journal of Cardiovascular Drugs, 2008 - ingentaconnect.com Most hypertensive patients will require more than one antihypertensive drug to
lower BP below target levels. Abstract The combination of diuretics with
renin-angiotensin system (RAS) antagonists offers several advantages to ... Cited by 5 - Related articles - BL Direct - All 6 versions
BF Palmer, ASA Naderi - Journal of the American Society of Hypertension, 2007 - Elsevier Thaizides are effective agents in lowering blood pressure when used as
monotherapy and they provide additive blood pressure lowering effects when
combined with many other antihypertensive drugs. The seventh Joint National ... Cited by 1 - Related articles - All 3 versions
- ►cell.com JC Koster, HT Kurata, D Enkvetchakul, CG … - Biophysical Journal, 2008 - Elsevier ATP-sensitive K + -channels link metabolism and excitability in neurons,
myocytes, and pancreatic islets. Mutations in the pore-forming subunit (Kir6.2;
KCNJ11) cause neonatal diabetes, developmental delay, and epilepsy by ... Related articles - All 7 versions
CA Girard, FT Wunderlich, K Shimomura, S … - The Journal of Clinical Investigation, 2009 - ukpmc.ac.uk Neonatal diabetes is a rare monogenic form of diabetes that usually presents
within the first six months of life. It is commonly caused by gain-of-function
mutations in the genes encoding the Kir6.2 and SUR1 subunits of the ... Related articles
G Reboldi, G Gentile, F Angeli, P Verdecchia - Vascular Health and Risk Management, 2009 - pubmedcentral.nih.gov The diabetes epidemic continues to grow unabated, with a staggering toll in
micro- and macrovascular complications, disability, and death. Diabetes causes a
two- to fourfold increase in the risk of cardiovascular disease, and ... Cited by 1 - Related articles - All 6 versions