- ►cjb.net E Planel, Y Tatebayashi, T Miyasaka, L Liu, … - Journal of Neuroscience, 2007 - neuro.cjb.net Hyperphosphorylated tau is the major component of paired helical filaments in
neurofibrillary tangles found in Alzheimer's disease (AD) brains, and tau
hyperphosphorylation is thought to be a critical event in the pathogenesis ... Cited by 22 - Related articles - All 4 versions
- ►aspetjournals.org I Vulih-Shultzman, A Pinhasov, S Mandel, N … - Journal of Pharmacology and Experimental …, 2007 - ASPET Activity-dependent neuroprotective protein (ADNP) differentially interacts with
chromatin to regulate essential genes. Because complete ADNP deficiency is
embryonic lethal, the outcome of partial ADNP deficiency was examined. ADNP ... Cited by 12 - Related articles - All 4 versions
JR Babu, ML Seibenhener, J Peng, AL Strom … - J Neurochem, 2008 - interscience.wiley.com The signaling adapter p62 plays a coordinating role in mediating phosphorylation
and ubiquitin-dependent trafficking of interacting proteins. However, there is
little known about the physiologic role of this protein in brain. Here, we ... Cited by 10 - Related articles - All 4 versions
- ►irb.hr [PDF] M Salkovic-Petrisic, S Hoyer - JOURNAL OF NEURAL TRANSMISSION …, 2007 - Springer J Neural Transm (2007) [Suppl 72]: 217–233 # Springer-Verlag 2007 Printed in
Austria ... Central insulin resistance as a trigger for sporadic Alzheimer-like
pathology: an experimental approach ... M. Salkovic-Petrisic 1 , S. Hoyer 2 Cited by 11 - Related articles - BL Direct - All 7 versions
- ►alzheimer-forschung.de [PDF] V Meske, F Albert, TG Ohm - Journal of Biological Chemistry, 2008 - ASBMB Tau is an important microtubule-stabilizing protein in neurons. In its
hyperphosphorylated form, Tau protein loses its ability to bind to microtubules
and then accumulates and is part of pathological lesions characterizing ... Cited by 8 - Related articles - All 5 versions
I Torres-Aleman - Endocrinology, 2008 - Endocrine Soc It is lay knowledge now that Alzheimer's dementia (AD) is one of the most
devastating diseases afflicting our societies. A major thrust in search for a
cure has relied in the development of animal models of the disease. Thanks ... Cited by 4 - Related articles - All 5 versions
- ►j-talk.com FG De Felice, MNN Vieira, TR Bomfim, H … - Proceedings of the National Academy of Sciences, 2009 - National Acad Sciences Synapse deterioration underlying severe memory loss in early Alzheimer's disease
(AD) is thought to be caused by soluble amyloid beta (Aβ) oligomers.
Mechanistically, soluble Aβ oligomers, also referred to as Aβ-derived ... Cited by 4 - Related articles - All 10 versions
RJ Gomes, CAM de Oliveira, C Ribeiro, CSA … - Hippocampus, 2009 - interscience.wiley.com The present study investigated the role of swimming training on cerebral
metabolism and hippocampus concentrations of insulin and IGF-1 in diabetic rats.
Wistar rats were divided in sedentary control (SC), trained control (TC), ... Related articles - All 2 versions
AAF Sima - diabetes - Springer This review will compare longitudinally the cognitive deficits and associated
metabolic and structural abnormalities in two models with spontaneous onset of
type 1 and type 2 diabetes, respectively. From these studies it is becoming ... Related articles - All 2 versions
SA Wrighten, GG Piroli, CA Grillo, LP Reagan - BBA-Molecular Basis of Disease, 2009 - Elsevier Central nervous system (CNS) complications resulting from diabetes is a problem
that is gaining more acceptance and attention. Recent evidence suggests
morphological, electrophysiological and cognitive changes, often observed ... Related articles - All 2 versions
