L Bjorkhaug, J Molnes, O Sovik, PR Njolstad, … - Journal of biological chemistry, 2007 - ASBMB Running Title: Glucokinase activation and proteasomal degradation ... 1 Address
correspondence to: Torgeir Flatmark, Department of Biomedicine, University of
Bergen, Jonas Lies vei 91, 5009 Bergen, Norway. Tel.: +47 55586428; Fax:+ ... Cited by 8 - Related articles - All 6 versions
- ►nih.gov TS Leak, JC Mychaleckyj, SG Smith, KL … - Human genetics, 2008 - Springer Abstract Previously, we performed a genome scan for type 2 diabetes (T2DM) using
638 African-American (AA) affected sibling pairs from 247 families;
non-parametric linkage analysis suggested evidence of linkage at 6q24–27 ... Cited by 2 - Related articles - All 6 versions
S Wing - BMC biochemistry, 2008 - biomedcentral.com Type 2 diabetes is caused by defects in both insulin signaling and insulin
secretion. Though the role of the ubiquitin proteasome system (UPS) in the
pathogenesis of type 2 diabetes remains largely unexplored, the few ... Cited by 2 - Related articles - Cached - All 6 versions
T Hartley, J Brumell, A Volchuk - American Journal of Physiology- Endocrinology And …, 2009 - Am Physiological Soc Protein degradation in eukaryotic cells is mediated primarily by the
ubiquitin-proteasome system and autophagy. Turnover of protein aggregates and
other cytoplasmic components, including organelles, is another function ... Cited by 1 - Related articles - All 3 versions
C IONESCU-TÎRGOVIŞTE, C GUJA - paulescu.ro Based on our clinical and epidemiological data, we have sustained for a long
time the unitary character of the various phenotypes of the diabetic syndrome.
In this paper, we add several arguments sustaining that the unitary ... Related articles - View as HTML - All 2 versions
Z Ma - diss.kib.ki.se This thesis attempts to further clarify mechanisms behind the negative effects
of over- stimulation and the beneficial effect of ß-cell rest. For this purpose
diazoxide - opener of K+-ATP channels, which reversibly inhibits ... Related articles