G Twig, B Hyde, OS Shirihai - BBA-Bioenergetics, 2008 - Elsevier The mitochondrial life cycle consists of frequent fusion and fission events. Ample experimental
and clinical data demonstrate that inhibition of either fusion or fission results in deterioration
of mitochondrial bioenergetics. While fusion may benefit mitochondrial function by ... Cited by 25 - Related articles - All 2 versions
- ►nih.gov DJ Taatjes, BE Sobel, RC Budd - Histochemistry and Cell Biology, 2008 - Springer Page 1. Histochem Cell Biol (2008) 129:33–43 DOI 10.1007/s00418-007-0356-9 123
REVIEW Morphological and cytochemical determination of cell death by apoptosis
Douglas J. Taatjes · Burton E. Sobel · Ralph C. Budd Accepted ... Cited by 13 - Related articles - BL Direct - All 7 versions
- ►nih.gov AA Gerencser, DG Nicholls - Biophysical Journal, 2008 - Elsevier Impaired transport of mitochondria, in dendrites and axons of neurons, and bioenergetic deficit
are increasingly recognized to be of pathological importance in neurodegenerative
diseases. To study the relationship between transport and bioenergetics, we have ... Cited by 6 - Related articles - All 9 versions
J Bereiter-Hahn, M Vöth, S Mai, M Jendrach - Biotechnol J, 2008 - interscience.wiley.com Mitochondrial components are continuously distributed throughout the whole chondriome of
a cell by fusion and fission. Thus, a single mitochondrion represents a transient fraction of the
chondriome. Mitochondrial dynamics are responsible for intracellular distribution and ... Cited by 5 - Related articles - All 2 versions
- ►physiology.org SC Gunawardana, WS Head, DW … - American Journal of …, 2008 - Am Physiological Soc Dimethyl amiloride (DMA) enhances insulin secretion in the pancreatic β-cell. DMA also enhances
time-dependent potentiation (TDP) and enables TDP to occur in situations where it is normally
absent. As we have demonstrated before, these effects are mediated in part through ... Cited by 1 - Related articles - All 3 versions
- ►ccf.org [PDF] T Koeck, JA Corbett, JW Crabb, DJ Stuehr, KS … - Archives of Biochemistry …, 2009 - Elsevier Hyperglycemia, key factor of the pre-diabetic and diabetic pathology, is associated with cellular
oxidative stress that promotes oxidative protein modifications. We report that protein nitration
is responsive to changes in glucose concentrations in islets of Langerhans and ... Cited by 1 - Related articles - All 6 versions
- ►jneurosci.org P Weisova, CG Concannon, M Devocelle, JHM … - Journal of …, 2009 - Soc Neuroscience Ischemic and excitotoxic events within the brain result in rapid and often unfavorable depletions
in neuronal energy levels. Here, we investigated the signaling pathways activated in response
to the energetic stress created by transient glutamate excitation in cerebellar granule ... Cited by 1 - Related articles - All 4 versions
G Szabadkai, MR Duchen - Apoptosis - Springer Abstract Mitochondrial dysfunction plays a role in the pathogenesis of a wide range of diseases
that involve dis- ordered cellular fuel metabolism and survival/death path- ways, including neurodegenerative
diseases, cancer and diabetes. Cytokine, virus recognition and cellular stress pathways ... Related articles - All 2 versions
NN Danial - Oncogene, 2008 - nature.com The BH3-only pro-apoptotic proteins are upstream sensors of cellular damage that selectively
respond to specific, proximal death and survival signals. Genetic models and biochemical studies
indicate that these molecules are latent killers until activated through transcriptional or ... Related articles - All 3 versions
S Liu, T Okada, A Assmann, J Soto, CW Liew… - PLoS ONE, 2009 - plosone.org Insulin/IGF-I signaling regulates the metabolism of most mammalian tissues including pancreatic
islets. To dissect the mechanisms linking insulin signaling with mitochondrial function, we first
identified a mitochondria-tethering complex in β-cells that included glucokinase (GK), and ... Cached
