- ►endocrinology-journals.org I Quesada, E Tuduri, C Ripoll, A Nadal - Journal of Endocrinology, 2008 - Soc Endocrinology The secretion of glucagon by pancreatic -cells plays a critical role in the
regulation of glycaemia. This hormone counteracts hypoglycaemia and opposes
insulin actions by stimulating hepatic glucose synthesis and mobilization, ... Cited by 8 - Related articles - All 3 versions
- ►nih.gov L Eliasson, F Abdulkader, M Braun, J … - The Journal of Physiology, 2008 - Physiological Soc Pancreatic β-cells secrete insulin by Ca 2+ -dependent exocytosis of secretory
granules. β-cell exocytosis involves SNARE (soluble NSF-attachment protein
receptor) proteins similar to those controlling neurotransmitter release ... Cited by 8 - Related articles - All 6 versions
- ►physiology.org P Bansal, Q Wang - American Journal of Physiology- Endocrinology And …, 2008 - Am Physiological Soc Glucose homeostasis is regulated primarily by the opposing actions of insulin
and glucagon, hormones that are secreted by pancreatic islets from β-cells and
-cells, respectively. Insulin secretion is increased in response to ... Cited by 6 - Related articles - All 3 versions
- ►diabetesjournals.org AC Hauge-Evans, AJ King, D Carmignac, CC … - Diabetes, 2009 - Am Diabetes Assoc OBJECTIVE— Somatostatin (SST) is secreted by islet δ-cells and by extraislet
neuroendocrine cells. SST receptors have been identified on α- and β-cells,
and exogenous SST inhibits insulin and glucagon secretion, consistent with ... Cited by 5 - Related articles - All 8 versions
- ►cell.com P Rorsman, SA Salehi, F Abdulkader, M … - Trends in Endocrinology & Metabolism, 2008 - Elsevier Glucagon, secreted by the α-cells of the pancreatic islets, is the most
important glucose-increasing hormone of the body. The precise regulation of
glucagon release remains incompletely defined but has been proposed to ... Cited by 4 - Related articles - All 5 versions
N Tyagi, D Lominadze, W Gillespie, KS … - Clinical Chemical Laboratory Medicine, 2007 - reference-global.com Background: g-Aminobutyric acid (GABA) is a known inhibitory neurotransmitter in
the mammalian central nervous system, and homocysteine (Hcy) behaves as an
antagonist for GABA A receptor. Although the properties and functions of ... Cited by 2 - Related articles - BL Direct - All 4 versions
- ►diabetesjournals.org N Quoix, R Cheng-Xue, L Mattart, Z Zeinoun, … - Diabetes, 2009 - Am Diabetes Assoc RESULTS—In 0.5 mmol/l glucose, [Ca 2+ ] c oscillated in some -cells and was
basal in the others. Increasing glucose to 15 mmol/l decreased [Ca 2+ ] c by 30%
in oscillating cells and was ineffective in the others. -Cell I KATP was ... Cited by 1 - Related articles - All 3 versions
GA Rutter - Diabetes, 2009 - Am Diabetes Assoc Despite being a subject under investigation for more than 35 years (6), just how
the effects of glucose are achieved at the level of individual α-cells is still
disputed and has become an area of vigorous research in recent times ... Related articles - All 3 versions
SZ Young, A Bordey - Physiology, 2009 - Am Physiological Soc Aside from traditional neurotransmission and regulation of secretion, -amino
butyric acid (GABA) through GABA A receptors negatively regulates proliferation
of pluripotent and neural stem cells in embryonic and adult tissue. There ... Related articles - All 4 versions