P Boor, K Sebekova, T Ostendorf, J Floege - Nephrology Dialysis Transplantation, 2007 - ERA-EDTA Mechanisms contributing to tubulointerstitial injury and tubular atrophy include
glomerular proteinuria, chronic hypoxia, misdirected glomerular ultrafiltration,
tubular protein leakage and direct toxic insults of eg drugs (reviewed in ... Cited by 10 - Related articles - BL Direct - All 6 versions
PW Anderson, JB McGill, KR Tuttle - Current Opinion in Nephrology and Hypertension, 2007 - journals.lww.com Recent findings: Ruboxistaurin mesylate is a bisindolylmaleimide that
specifically inhibits the β-isoform of protein kinase C. In animal models of
diabetic nephropathy, ruboxistaurin normalized glomerular hyperfiltration, ... Cited by 6 - Related articles - BL Direct - All 7 versions
AK Goestemeyer, J Marks, SK Srai, ES … - Diabetologia, 2007 - Springer Abstract Aims/hypothesis GLUT2 is the main renal glucose transport- er
upregulated by hyperglycaemia, when it becomes detect- able at the brush border
membrane (BBM). Since glucose- induced protein kinase C (PKC) activation in ... Cited by 6 - Related articles - BL Direct - All 4 versions
- ►critcaremed.com C Chatziantoniou, JC Dussaule - Current Opinion in Nephrology and Hypertension, 2008 - journals.lww.com Purpose of review: Recent evidence indicates that the progression of renal
fibrosis is a reversible process in experimental models. This review summarizes
the new insights concerning the mechanisms of progression and regression of ... Cited by 5 - Related articles - BL Direct - All 16 versions
- ►oxfordjournals.org M Meier, J Menne, JK Park, H Haller - Nephrology Dialysis Transplantation, 2007 - ERA-EDTA Diabetic nephropathy is characterized by early vascular dysfunction and
increasing matrix accumulation in the kidney, eventually leading to proteinuria,
glomerulosclerosis and interstitial fibrosis [1]. Over the last decade, our ... Cited by 4 - Related articles - BL Direct - All 2 versions
EN Lavrentyev, KU Malik - American Journal of Physiology- Heart and Circulatory …, 2009 - Am Physiological Soc In rat diabetic animal models, ANG(1-7) treatment prevents the development of
cardiovascular complications. Angiotensin-converting enzyme (ACE)2 is a major
ANG(1-7)-generating enzyme in vascular smooth muscle cells (VSMCs), and its ... Cited by 4 - Related articles - All 3 versions
M Meier, J Menne, H Haller - Diabetologia, 2009 - Springer Abstract The protein kinase C (PKC) superfamily com- prises proteins that are
activated in response to various pathogenic stimuli in the diabetic state.
Hyperglycaemia is the predominant stimulus that induces the activation of ... Cited by 3 - Related articles - All 2 versions
SI Lee, SH Oh, KY Park, BH Park, JS Kim, SD … - Journal of Medicinal Food, 2009 - liebertonline.com Sang-Il Lee, 1 Sung-Hee Oh, 2 Kun-Young Park, 3 Bum-Ho Park, 4 Jeong-Sook Kim, 1
and Soon-Dong Kim 4 ... 1 Department of Food, Nutrition and Cookery, Keimyung
College, Daegu; ... Art, Pohang College, Gyeongbuk; 3 Department of Food ... Cited by 1 - Related articles - All 3 versions
NPJ Vogtländer, HJ Visch, MAH Bakker, JHM … - PloS one, 2009 - pubmedcentral.nih.gov Conditionally immortalized podocytes were exposed in vitro to antibodies to
α-dystroglycan, and to fibronectin, biglycan, laminin and agrin. Intracellular
calcium fluxes, phosphorylation of β-dystroglycan and podocyte ... Cited by 1 - Related articles - All 5 versions
