- ►endojournals.org D Scheuner, RJ Kaufman - Endocrine Reviews, 2008 - Endocrine Soc The endoplasmic reticulum (ER) is the entry site into the secretory pathway for
newly synthesized proteins destined for the cell surface or released into the
extracellular milieu. The study of protein folding and trafficking within ... Cited by 38 - Related articles - BL Direct - All 5 versions
CL Birmingham, V Canadien, NA Kaniuk, BE … - DNA - microbiology.ucsf.edu Listeria monocytogenes is an intracellular bacterial pathogen that replicates
rapidly in the cytosol of host cells during acute infec- tion1. Surprisingly,
these bacteria were found to occupy vacuoles in liver granuloma macrophages ... Cited by 30 - Related articles - View as HTML - BL Direct - All 12 versions
A Salehi, SM Abaraviciene, J Jimenez- … - PLoS One, 2008 - pubmedcentral.nih.gov We show now that islets of type 2 spontaneous diabetes in GK rats display
excessive NO generation associated with abnormal iNOS expression in insulin and
glucagon cells, increased ncNOS activity, impaired glucose-stimulated ... Cited by 10 - Related articles - All 8 versions
A Tomas, P Meda, R Regazzi, JE Pessin, PA … - Traffic, 2008 - interscience.wiley.com Munc 18-1 is a member of the Sec/Munc family of syntaxin-binding proteins known
to bind to the plasma membrane Q-SNARE syntaxin1 and whose precise role in
regulated exocytosis remains controversial. Here, we show that Munc 18-1 ... Cited by 9 - Related articles - BL Direct - All 4 versions
NM Wolters, JP MacKeigan - Cell Death & Differentiation, 2008 - nature.com RNA interference (RNAi) has emerged as one of the most powerful tools for
functionally characterizing large sets of genomic data. Capabilities of RNAi
place it at the forefront of high-throughput screens, which are able to ... Cited by 5 - Related articles - BL Direct - All 3 versions
- ►physiology.org MA Kiraly, HE Bates, NA Kaniuk, JTY Yue, JH … - American Journal of Physiology- Endocrinology And …, 2008 - Am Physiological Soc Exercise improves glucose tolerance in obese rodent models and humans; however,
effects with respect to mechanisms of β-cell compensation remain unexplained.
We examined exercise's effects during the progression of hyperglycemia in ... Cited by 4 - Related articles - BL Direct - All 3 versions
S Wing - BMC biochemistry, 2008 - biomedcentral.com Type 2 diabetes is caused by defects in both insulin signaling and insulin
secretion. Though the role of the ubiquitin proteasome system (UPS) in the
pathogenesis of type 2 diabetes remains largely unexplored, the few ... Cited by 2 - Related articles - Cached - All 6 versions
L Zhang, E Lai, T Teodoro, A Volchuk - Journal of Biological Chemistry, 2009 - ASBMB Chronic hyperglycemia contributes to pancreatic β-cell dysfunction during the
development of type 2 diabetes. Treatment of pancreatic β-cells with prolonged
high glucose concentrations has been shown to reduce insulin promoter ... Cited by 1 - Related articles - All 3 versions
RA Ribeiro, ML Bonfleur, AG Amaral, EC … - Diabetes/Metabolism Reviews - interscience.wiley.com It is also possible that your web browser is not configured or not able to
display style sheets. In this case, although the visual presentation will be
degraded, the site should continue to be functional. We recommend using the ... Cited by 1 - Related articles - All 2 versions
