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Scholar Results 1 - 10 of about 41 citing Chen: High glucose impairs early and late endothelial progenitor cells by modifying nitric.... (0.13 sec) 

Aging and disease as modifiers of efficacy of cell therapy

- ahajournals.org
S Dimmeler, A Leri - Circulation research, 2008 - Am Heart Assoc
Cell therapy is a promising option for treating ischemic diseases and heart
failure. Adult stem and progenitor cells from various sources have
experimentally been shown to augment the functional recovery after ...
Cited by 23 - Related articles - All 11 versions

Phosphoinositide 3-Kinase {gamma} Gene Knockout Impairs Postischemic …

- ahajournals.org
P Madeddu, N Kraenkel, LS Barcelos, M … - Arteriosclerosis, Thrombosis, and Vascular Biology, 2008 - Am Heart Assoc
Methods and Results— Unilateral limb ischemia was induced in mice lacking the
PI3K gene (PI3K –/– ) or expressing a catalytically inactive mutant (PI3K
KD/KD ) and wild-type controls (WT). Capillarization and arteriogenesis ...
Cited by 16 - Related articles - All 7 versions

In vitro hyperglycemia or a diabetic intrauterine environment reduces neonatal endothelial …


DA Ingram, IZ Lien, LE Mead, M Estes, DN … - Diabetes, 2008 - Am Diabetes Assoc
OBJECTIVE—Emerging data demonstrate that maternal diabetes has long-term
health consequences for offspring, including the development of hypertension. In
adults, circulating endothelial progenitor cells (EPCs) participate in ...
Cited by 12 - Related articles - BL Direct - All 4 versions

Advanced glycation of fibronectin impairs vascular repair by endothelial progenitor cells: …

- iovs.org
AD Bhatwadekar, JV Glenn, G Li, TM Curtis, TA … - Investigative Ophthalmology & Visual Science, 2008 - ARVO
RESULTS. EPC attachment and spreading on AGE-FN was reduced compared with
control cells (P < 0.05–0.01) but was significantly restored by pretreatment
with Arg-Gly-Asp (RGD). Chemotaxis of EPCs was abolished on AGE-FN but was ...
Cited by 10 - Related articles - BL Direct - All 2 versions

High glucose downregulates endothelial progenitor cell number via SIRT1

- proteomicslabs.com [PDF] 
ML Balestrieri, M Rienzo, F Felice, R … - BBA-Proteins and Proteomics, 2008 - Elsevier
Increasing evidence indicates that mammalian SIRT1 mediates calorie restriction
and influences lifespan regulating a number of biological molecules such as
FoxO1. SIRT1 controls the angiogenic activity of endothelial cells via ...
Cited by 8 - Related articles - All 10 versions

Targeting phosphoinositide 3-kinase gamma to fight inflammation and more


L Barberis, E Hirsch - THROMBOSIS AND HAEMOSTASIS-STUTTGART-, 2008 - schattauer.de
Summary The family of class I phosphoinositide-3-kinase (PI3K) is composed of
four lipid kinases involved at multiple levels in in- nate and adaptive immune
responses. Class I PI3Ks are divided into two subclasses, IA and IB, ...
Cited by 7 - Related articles - BL Direct - All 5 versions

Microvascular endothelial cell heterogeneity: general concepts and pharmacological …


E Langenkamp, G Molema - Cell and Tissue Research, 2009 - Springer
Abstract Microvascular endothelial cells display a large degree of heterogeneity
in function depending on their location in the vascular tree. The existence of
organ- specific, microvascular-bed-specific, and even intravascular ...
Cited by 6 - Related articles - All 3 versions

Diabetes and vessel wall remodelling: from mechanistic insights to regenerative therapies

- oxfordjournals.org
G Spinetti, N Kraenkel, C Emanueli, P … - Cardiovascular Research, 2008 - cardiovascres.oxfordjournals.org
Over the past two decades, extensive research has focused on arterial
remodelling in both physiological and pathological ageing. The concept now
describes the growth as well as the rearrangement of cellular components ...
Cited by 6 - Related articles - All 4 versions

Glucose induces apoptosis of cardiomyocytes via microRNA-1 and IGF-1


XY Yu, YH Song, YJ Geng, QX Lin, ZX Shan, … - Biochemical and Biophysical Research …, 2008 - Elsevier
Glucose toxicity is an important initiator of cardiovascular disease,
contributing to the development of cardiomyocyte death and diabetic
complications. The present study investigated whether high glucose state ...
Cited by 6 - Related articles - All 4 versions

p66ShcA modulates oxidative stress and survival of endothelial progenitor cells in response …

- cardiovascres.org
V Di Stefano, C Cencioni, G Zaccagnini, A … - Cardiovascular Research, 2009 - cardiovascres.oxfordjournals.org
Aims: A close relationship exists between hyperglycaemia, oxidative stress, and
diabetic complications. In fact, high glucose (HG) determines the overproduction
of reactive oxygen species (ROS) by the mitochondria. p66 ShcA is a gene ...
Cited by 4 - Related articles - All 4 versions


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