BM Lyttle, J Li, M Krishnamurthy, F Fellows, MB … - Diabetologia, 2008 - Springer Abstract Aims/hypothesis Morphological changes that occur during pancreatic endocrine cell
differentiation have been shown in rodent systems to be dependent on sequential alterations
in transcription factor expression. However, similar data for humans have been limited. ... Cited by 11 - Related articles - BL Direct - All 2 versions
- ►prsjournal.net P Khatri, MM Sarwal - Current Opinion in Organ Transplantation, 2009 - journals.lww.com Recent findings: Microarrays have been useful in identifying potential biomarkers for chronic
rejection in peripheral blood mononuclear cells, novel pathways for induction of tolerance, and
genes involved in protecting the graft from the host immune system. Microarray analysis ... Cited by 1 - Related articles - All 6 versions
M Cananzi, A Atala, P De Coppi - Reproductive BioMedicine Online, 2009 - ingentaconnect.com Paolo De Coppi is a Clinical Senior Lecturer and Consultant in Paediatric Surgery at the UCL
Institute of Child Health and Great Ormond Street Hospital, London, UK. His clinical training and
PhD in tissue engineering and cell transplantation (2002) were undertaken at the ... Cited by 2 - Related articles - All 2 versions
MR Hammerman - Transplant Immunology, 2009 - Elsevier Lack of donor availability limits the number of human donor organs. The need for host immunosuppression
complicates transplantation procedures. It is possible to 'grow' new pancreatic tissue or kidneys
in situ via xenotransplantation of organ primordia from animal embryos (organogenesis of ... Cited by 1 - Related articles - All 2 versions
G Fousteri, M von Herrath - DIABETES, 2008 - Am Diabetes Assoc T ype 1 diabetes results from immune-mediated destruction of insulin-producing -cells of the
pancreas. The disease manifests itself symptom- atically when the resident -cells become unable
to maintain normoglycemia. In human insulitis, mainly CD8 lymphocytes and B-cells, but ... Related articles
G Fousteri, M von Herrath - Diabetes, 2008 - Am Diabetes Assoc Type 1 diabetes results from immune-mediated destruction of insulin-producing β-cells of the
pancreas. The disease manifests itself symptomatically when the resident β-cells become unable
to maintain normoglycemia. In human insulitis, mainly CD8 lymphocytes and B-cells, but ... Related articles - BL Direct - All 3 versions
BE Tuch, K Kannangara - Drug Discovery Today: Therapeutic Strategies, 2009 - Elsevier Replacing the 350 million β cells destroyed in people with type 1 diabetes has traditionally been
sought by transplanting human pancreatic tissue obtained after death. Alternative sources of
exogenous surrogate β cells being examined include stem cells, fetal pancreas and cells ... Related articles