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Scholar Results 1 - 7 of 7 citing Deering: Methyltransferase Set7/9 maintains transcription and euchromatin structure at islet-enri.... (0.08 sec) 

Sleeping Beauty, Awake! Regulation of Insulin Gene Expression by Methylation of Histone …


DR Cavener - Diabetes, 2009 - Am Diabetes Assoc
In all cell types of the body, save one, the insulin gene is quiescent and is
ensconced in a compact and protected environment of chromatin. How the insulin
gene is coaxed out of its chromatin slumber in the pancreatic β-cell is ...
Cited by 1 - Related articles - All 4 versions

Peroxisome Proliferator-Activated Receptor {gamma} Activation Restores Islet Function in …

- nih.gov
C Evans-Molina, RD Robbins, T Kono, SA … - Molecular and Cellular Biology, 2009 - Am Soc Microbiol
The nuclear receptor peroxisome proliferator-activated receptor (PPAR- ) is an
important target in diabetes therapy, but its direct role, if any, in the
restoration of islet function has remained controversial. To identify ...
Cited by 1 - Related articles - All 4 versions

[PDF] SET7/9 mediated methylation of non-histone proteins in mammalian cells


S Pradhan, HG Chin, PO Estčve, SE … - Epigenetics, 2009 - mcdb.ucla.edu
Post-translational modification (PTM) of histones, such as methylation,
acetylation, phosphorylation, ADP-ribosylation and ubiquitination are shown to
play important regulatory roles in defining the status of eukaryotic gene ...
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The Nucleosome Binding Protein HMGN3 Modulates the Transcription Profile of Pancreatic { …


T Ueda, T Furusawa, T Kurahashi, L … - Molecular and Cellular Biology, 2009 - Am Soc Microbiol
Improper glucose-stimulated insulin secretion from pancreatic β cells is a
major factor in the onset of type 2 diabetes. We now report that HMGN3, a
nuclear protein that binds to nucleosomes and affects chromatin function, ...
Related articles - All 3 versions

Epigenetics: deciphering how environmental factors may modify autoimmune type 1 …


AJ MacFarlane, A Strom, FW Scott - Mammalian Genome - Springer
Abstract Type 1 diabetes (T1D) is an autoimmune dis- ease that has increased
two- to threefold over the past half century by as yet unknown means. It is
generally accepted that T1D is the result of gene–environment ...
Related articles - All 2 versions

[PDF] Proinsulin As The Possible Key In The Pathogenesis Of Type 1 Diabetes


C Ionescu-Tîrgoviºte - Acta Endocrinologica (Buc), 2009 - 82.76.72.38
Abstract Based on epidemiological, clinic, biochemical and hormonal data (both
personal and international), our personal view is that natural history of
autoimmune type 1 diabetes could include the following stages: 1) The ...
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REGULATION OF THE L-TYPE PYRUVATE KINASE GENE BY GLUCOSE AND cAMP IN …

- pitt.edu [PDF] 
S Burke, 2009 - etd.library.pitt.edu
Page 1. REGULATION OF THE L-TYPE PYRUVATE KINASE GENE BY GLUCOSE AND cAMP IN
ISLET β CELLS by Susan Burke BSc, University College Cork, 2004 ...
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