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Scholar Results 1 - 3 of 3 citing Funai: Inhibition of contraction-stimulated AMP-activated protein kinase inhibits contraction-sti.... (0.06 sec) 

Increased AS160 phosphorylation, but not TBC1D1 phosphorylation, with increased …


K Funai, GG Schweitzer, N Sharma, M … - American Journal of Physiology- Endocrinology And …, 2009 - Am Physiological Soc
A single exercise bout can increase insulin-independent glucose transport
immediately postexercise and insulin-dependent glucose transport (GT) for
several hours postexercise. Akt substrate of 160 kDa (AS160) and TBC1D1 are ...
Cited by 3 - Related articles - All 3 versions

Exercise and Insulin: Convergence or Divergence at AS160 and TBC1D1?


GD Cartee, K Funai - Exercise and Sport Sciences Reviews, 2009 - journals.lww.com
CARTEE, GD and K. FUNAI. Exercise and Insulin: Convergence or Divergence atAS160
and TBC1D1? Exerc. Sport Sci. Rev., Vol. 37, No. 4, pp. 188Y195, 2009. Akt
substrate of 160 kDa (called AS160 or TBC1D4) and TBC1D1, Rab ...
Cited by 1 - Related articles

Restoring AS160 phosphorylation rescues skeletal muscle insulin resistance and fatty acid …


H Alkhateeb, A Chabowski, JFC Glatz, B Gurd … - American Journal of Physiology- Endocrinology And …, 2009 - Am Physiological Soc
We examined whether AICAR or leptin rapidly rescued skeletal muscle insulin
resistance via increased palmitate oxidation, reductions in intramuscular
lipids, and/or restoration of insulin-stimulated AS60 phosphorylation. ...
Related articles - All 3 versions


 


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