- ►ahajournals.org [PDF] O Sitbon, M Humbert, X Jais, V Ioos, AM … - Circulation, 2005 - Am Heart Assoc Kluwer Health, 351 West Camden Street, Baltimore, MD 21202-2436. Phone:
410-528-4050. Permissions: Permissions & Rights Desk, Lippincott Williams &
Wilkins, a division of Wolters ... Long-Term Response to Calcium Channel ... Cited by 290 - Related articles - All 9 versions
O Sitbon, F Brenot, A Denjean, A Bergeron, F … - Reanimation Urgences, 1996 - Elsevier To investigate the capacity of the pulmonary vascular bed to acutely vasodilate,
we examined in 35 consecutive patients with primary pulmonary hypertension
(PPH), the hemodynamic effects of incremental inhalation periods of an ... Cited by 248 - Related articles - BL Direct - All 8 versions
- ►ahajournals.org [PDF] HI Palevsky, BL Schloo, GG Pietra, KT Weber, … - Circulation, 1989 - Am Heart Assoc Harold I. Palevsky, MD, Betsy L. Schloo, MD, Giuseppe G. Pietra, MD, Karl T.
Weber, MD, Joseph S. Janicki, PhD, Eben Rubin, MD, and Alfred P. Fishman, MD ... The use of pharmacologic agents in the treatment of pulmonary ... Cited by 161 - Related articles - All 5 versions
A Mebazaa, P Karpati, E Renaud, L Algotsson - Intensive care medicine, 2004 - Springer Abstract The right ventricle (RV) provides sustained low-pressure per- fusion of
the pulmonary vasculature, but is sensitive to changes in loading conditions and
intrinsic contractility. Factors that affect right ventricular preload, ... Cited by 111 - Related articles - BL Direct - All 4 versions
- ►nursing2007.com A Morelli, JL Teboul, SM Maggiore, A … - Critical care medicine, 2006 - journals.lww.com Objective: Acute respiratory distress syndrome (ARDS) is fre- quently associated
with increased pulmonary vascular resistance and thus with systolic load of the
right ventricle. We hypothesized that levosimendan, a new calcium ... Cited by 77 - Related articles - BL Direct - All 12 versions
I Adatia, S Perry, M Landzberg, P Moore, JE … - Journal of the American College of Cardiology, 1995 - Elsevier Results. In group I, inhaled nitric oxide decreased pulmonary artery pressure
from (mean ± SE) 71 ± 13 to 59 ± 10 mm Hg (p < 0.05), pulmonary vascular
resistance from 14.9 ± 3.8 to 7.6 ± 1.7 Um 2 (p < 0.05) and ... Cited by 77 - Related articles - BL Direct - All 6 versions
- ►nih.gov [PDF] JSR Gibbs, TW Higenbottam, SG Haworth, K … - Heart, 2001 - pubmedcentral.nih.gov Synopsis PRESENTATION AND INVESTIGATION 1. The symptoms of pulmonary
hypertension are relatively non-specific. Breathlessness is the most common. 2.
Where the diagnosis of pulmonary hyper- tension is suspected a ... Cited by 66 - Related articles - Library Search - All 4 versions
- ►onlinejacc.org AM Atz, I Adatia, JE Lock, DL Wessel - Journal of the American College of Cardiology, 1999 - Elsevier We compared the ability of inhaled nitric oxide (NO), oxygen (O 2 ) and nitric
oxide in oxygen (NO+O 2 ) to identify reactive pulmonary vasculature in
pulmonary hypertensive patients during acute vasodilator testing at cardiac ... Cited by 64 - Related articles - BL Direct - All 11 versions
MC PRAGER, CA CAULDWELL, NL ASCHER, … - Anesthesiology, 1992 - journals.lww.com Anesthesiology V 77, No 2, Aug 1992 CASE REPORTS 375 References 1. Vervloet D,
Nizankowska E, Arnaud A, Senft M, Charpin J: Ad- verse reactions to
suxamethonium and other muscle relaxants under general anesthesia. J ... Cited by 63 - Related articles - All 3 versions
