- ►nih.gov [PDF] RG Crystal - Journal of Clinical Investigation, 1990 - pubmedcentral.nih.gov Address reprint requests to Dr. Crystal, Pulmonary Branch, National Institutes of Health,Building
10, Room 6D03, Bethesda, MD 20892. Receivedfor publication 20 November 1989 and in revisedform 22
January 1990. Cited by 277 - Related articles - All 7 versions
- ►nih.gov [PDF] HA Jaffe, R Buhl, A Mastrangeli, KJ … - Journal of Clinical …, 1991 - pubmedcentral.nih.gov In the context of the central role of the alveolar macrophage in host defense of the respiratory
epithelial surface, and the abil- ity ofIFN-'yto activate mononuclear phagocytes, we have evalu-
ated strategies to use rIFN-'y to ... Cited by 89 - Related articles - All 8 versions
- ►171.66.122.149 RA Stockley, DL Bayley, I Unsal, LJ … - American journal of …, 2002 - 171.66.122.149 1-Antitrypsin (AAT) deficiency predisposes to bronchitis and emphysema associated with neutrophilic
airway inflammation. The efficacy of augmentation therapy has not been proven clinically or by
demonstrating an effect on airway ... Cited by 77 - Related articles - BL Direct - All 5 versions
- ►pnas.org [PDF] R Buhl, C Vogelmeier, M Critenden, RC … - Proceedings of the …, 1990 - National Acad Sciences Page 1. Proc. Nail. Acad. Sci. USA Vol. 87, pp. 4063-4067, June 1990 Medical Sciences Augmentation
of glutathione in the fluid lining the epithelium of the lower respiratory tract by directly
administering glutathione aerosol (lung/antioxidant/therapy/bronchoalveolar lavage) ... Cited by 64 - Related articles - All 7 versions
RJ Coakley, C Taggart, S O'Neill, NG … - The American journal of …, 2001 - journals.lww.com ABSTRACT: 1-antitrypsin (1AT) deficiency is a com- mon lethal hereditary disorder of white persons
of Eu- ropean descent. The condition is characterized by re- duced serum levels of 1AT, a 52-kDa
glycoprotein synthesized chiefly in ... Cited by 57 - Related articles - BL Direct - All 3 versions
- ►nih.gov [PDF] RC Hubbard, NG McElvaney, SE Sellers, … - Journal of Clinical …, 1989 - pubmedcentral.nih.gov Alpha 1-antitrypsin(alAT)I deficiency is a genetic disorder characterized by low plasma and lung
levels ofa lAT and the development of emphysema by the third to fourth decades (1, 2). The
deficiency of alAT in the lower ... Cited by 50 - Related articles - All 4 versions
- ►onlinejacc.org PJ Mateos-Cáceres, A García-Méndez, A Lopez … - Journal of the American …, 2004 - Elsevier The aim of this study was to analyze modifications in the plasma protein map during an acute
coronary syndrome (ACS) using proteomics. ... Proteomics is a new technology that allows the
detection and identification of ... Cited by 45 - Related articles - BL Direct - All 14 versions
SE Weinberger - The New England journal of medicine, 1993 - nejm.highwire.org Traditionally a discipline based on physiology for the functional assessment of patients with lung
disease, pulmonary medicine has been moving over the past decade into the realm of cellular and
molecular biology in an attempt to ... Cited by 42 - Related articles - All 9 versions
- ►bmj.com RA Sandhaus - British Medical Journal, 2004 - thorax.bmj.com Alpha-1-antitrypsin (AAT) deficiency is a genetic condition that increases the risk of developing
lung and liver disease, as well as other associated conditions. Most treatment of affected
individuals is not specifically ... Cited by 37 - Related articles - BL Direct - All 8 versions
