ME Mendelsohn, RH Karas - The New England journal of medicine, 1999 - nejm.highwire.org The incidence of cardiovascular disease differs significantly between men and
women, in part because of differences in risk factors and hormones. 1 The
incidence of atherosclerotic diseases is low in premenopausal women, rises ... Cited by 1857 - Related articles - BL Direct - All 15 versions
- ►onlinejacc.org MM Redfield, RJ Rodeheffer, SJ Jacobsen, DW … - Journal of the American College of Cardiology, 2002 - Am Coll Cardio Found BACKGROUND: Measurement of BNP concentration is approved for use in the
diagnosis of heart failure and may aid in the detection of left ventricular
dysfunction. Although BNP is approved for clinical use, there are few data ... Cited by 570 - Related articles - BL Direct - All 12 versions
- ►ahajournals.org MP Haynes, D Sinha, KS Russell, M Collinge, … - Circulation research, 2000 - Am Heart Assoc Abstract—17ß-Estradiol (E 2 ) is a rapid activator of endothelial nitric
oxide synthase (eNOS). The product of this activation event, NO, is a
fundamental determinant of cardiovascular homeostasis. We previously ... Cited by 323 - Related articles - BL Direct - All 5 versions
- ►oxfordjournals.org TS Mikkola, TB Clarkson - Cardiovascular research, 2002 - cardiovascres.oxfordjournals.org There is strong evidence from both human and nonhuman primate studies supporting
the conclusion that estrogen deficiency increases the progression of
atherosclerosis. More controversial is the conclusion that postmenopausal ... Cited by 255 - Related articles - All 8 versions
- ►endojournals.org KL Chambliss, PW Shaul - Endocrine reviews, 2002 - Endocrine Soc Over the past decade, clinical and basic research has demonstrated that estrogen
has a dramatic impact on the response to vascular injury and the development of
atherosclerosis. Further work has indicated that this is at least partially ... Cited by 227 - Related articles - BL Direct - All 8 versions
- ►jbc.org KS Russell, MP Haynes, T Caulin-Glaser, J … - Journal of Biological Chemistry, 2000 - ASBMB Estradiol (E 2 ) causes endothelium-dependent vasodilation, mediated, in part,
by enhanced nitric oxide (NO) release. We have previously shown that E 2
-induced activation of endothelial nitric oxide synthase (eNOS) reduces its ... Cited by 135 - Related articles - BL Direct - All 3 versions
MP Haynes, L Li, D Sinha, KS Russell, K … - Journal of Biological Chemistry, 2003 - ASBMB 17β-Estradiol activates endothelial nitric oxide synthase (eNOS), enhancing
nitric oxide (NO) release from endothelial cells via the phosphatidylinositol
3-kinase (PI3-kinase)/Akt pathway. The upstream regulators of this pathway ... Cited by 135 - Related articles - BL Direct - All 9 versions
- ►ahajournals.org A Saitta, D Altavilla, D Cucinotta, N Morabito, … - Arteriosclerosis, thrombosis, and vascular biology, 2001 - Am Heart Assoc Abstract—— The endothelium is thought to play an important role in the
genesis of atherosclerosis, and several lines of evidence suggest that the
effect of an intervention on endothelial function might predict its ... Cited by 130 - Related articles - BL Direct - All 9 versions
JB Park, EL Schiffrin - Journal of hypertension, 2001 - journals.lww.com Background: The heart and blood vessels are exposed to elevated blood pressure
(BP) in hypertensive patients, but their changes in response to BP or
non-hemodynamic stimuli may be different, and occur with different ... Cited by 127 - Related articles - BL Direct - All 5 versions
- ►annals.org [PDF] A Scuteri, AJG Bos, LJ Brant, L Talbot, EG … - Annals of internal medicine, 2001 - Am Coll Physicians Hormone Replacement Therapy and Longitudinal Changes in ... Angelo Scuteri, MD,
PhD; Angelo JG Bos, MD, PhD; Larry J. Brant, PhD; Laura Talbot, EdD, PhD; Edward
G. Lakatta, MD; and Jerome L. Fleg, MD ... Background: The incidence of ... Cited by 87 - Related articles - BL Direct - All 6 versions
