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Scholar Results 1 - 10 of about 51 citing Wormser: Hematogenous dissemination in early Lyme disease. (0.07 sec) 

The clinical assessment, treatment, and prevention of Lyme disease, human granulocytic …

- urgences-serveur.fr [PDF] 
GP Wormser, RJ Dattwyler, ED Shapiro, JJ … - Clinical Infectious Diseases, 2006 - UChicago Press
Featured in New York Times "Searching the Web for Flu Outbreaks" November 28,
2008 Using Internet Searches for Influenza Surveillance Philip M. Polgreen,
Yiling Chen, David M. Pennock, and Forrest D. Nelson One study, published ...
Cited by 208 - Related articles - BL Direct - All 40 versions

Diagnosis of Lyme borreliosis

- nih.gov
ME Aguero-Rosenfeld, G Wang, I Schwartz, … - Clinical Microbiology Reviews, 2005 - cmr.highwire.org
Clinical Microbiology Reviews, July 2005, p. 484-509, Vol. 18, No. 3
0893-8512/05/$08.00+0 doi:10.1128/CMR.18.3.484-509.2005 Copyright © 2005,
American Society for Microbiology. All Rights Reserved.
Cited by 139 - Related articles - BL Direct - All 19 versions

Early Lyme disease

- shouxi.net
GP Wormser - The New England journal of medicine, 2006 - nejm.highwire.org
Lyme disease is the most common tick-borne disease in the United States and Eu-
rope.1,2 In the United States, Lyme disease is most often acquired from the bite
of the Ixodes scapularis tick, with the spirochete Borrelia burgdorferi the ...
Cited by 37 - Related articles - BL Direct - All 7 versions

The critical role of the linear plasmid lp36 in the infectious cycle of Borrelia burgdorferi


MW Jewett, K Lawrence, AC Bestor, K Tilly, … - Molecular Microbiology, 2007 - pubmedcentral.nih.gov
Cited by 22 - Related articles - BL Direct - All 8 versions

Coordinated expression of Borrelia burgdorferi complement regulator-acquiring surface …

- nih.gov
T Bykowski, ME Woodman, AE Cooley, CA … - Infection and Immunity, 2007 - iai.highwire.org
The Lyme disease spirochete, Borrelia burgdorferi, is largely resistant to being
killed by its hosts' alternative complement activation pathway. One possible
resistance mechanism of these bacteria is to coat their surfaces with host ...
Cited by 14 - Related articles - BL Direct - All 8 versions

The pathogenesis of Lyme neuroborreliosis: from infection to inflammation


TA Rupprecht, U Koedel, V Fingerle, HW … - Molecular Medicine, 2008 - pubmedcentral.nih.gov
This review describes the current knowledge of the pathogenesis of acute Lyme
neuroborreliosis (LNB), from invasion to inflammation of the central nervous
system. Borrelia burgdorferi (Bb) enters the host through a tick bite on ...
Cited by 12 - Related articles - BL Direct - All 6 versions

Borrelia burgdorferi adhesins identified using in vivo phage display


S Antonara, RM Chafel, M LaFrance, J … - Molecular microbiology, 2007 - pubmedcentral.nih.gov
Borrelia burgdorferi, the agent of Lyme disease, disseminates from the site of
deposition by Ixodes ticks to cause systemic infection. Dissemination occurs
through the circulation and through tissue matrices, but the B. burgdorferi ...
Cited by 10 - Related articles - BL Direct - All 6 versions

Reinfection in patients with Lyme disease


RB Nadelman, GP Wormser, EJC Goldstein - Clinical Infectious Diseases, 2007 - UChicago Press
Featured in New York Times "Searching the Web for Flu Outbreaks" November 28,
2008 Using Internet Searches for Influenza Surveillance Philip M. Polgreen,
Yiling Chen, David M. Pennock, and Forrest D. Nelson One study, published ...
Cited by 8 - Related articles - All 4 versions

Lyme Disease—European Perspective


G Stanek, F Strle - Infectious Disease Clinics of North America, 2008 - Elsevier
Lyme disease or Lyme borreliosis in North America and in Europe are caused by
genospecies of the Borrelia burgdorferi sensu lato complex. Lyme borreliosis in
Europe, however, is somewhat different from the disease present in North ...
Cited by 9 - Related articles - All 8 versions

Marginal zone B-cell depletion impairs murine host defense against Borrelia burgdorferi …

- nih.gov
AA Belperron, CM Dailey, CJ Booth, LK … - Infection and immunity, 2007 - iai.highwire.org
Marginal zone B (MZB) cells are a B-cell subset that produces T-cell-independent
antibodies to blood-borne antigens. In this study, we examined the effects of
MZB cell depletion on the immune response to the Lyme disease spirochete ...
Cited by 7 - Related articles - BL Direct - All 8 versions


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