JC Gasson, RH Weisbart, SE Kaufman, SC … - Science, 1984 - sciencemag.org 286-bp JCV fragment functioned better in the sense orientation in fetal glial
cells, but in the antisense orientation in CV-1 cells. This is in contrast to
the SV40 and BKV enhancers, which are consistently more active in the sense ... Cited by 377 - Related articles - All 5 versions
V Nickeleit, T Klimkait, IF Binet, P Dalquen, … - New England Journal of Medicine, 2000 - content.nejm.org Results In all nine patients with BK virus nephropathy, BK virus DNA was
detected in the plasma at the time of the initial histologic diagnosis (a mean
[±SD] of 46±28 weeks after transplantation) and during the course of ... Cited by 308 - Related articles - BL Direct - All 5 versions
- ►asnjournals.org V Nickeleit, HH Hirsch, IF BINET, F Gudat, O … - Journal of the American Society of Nephrology, 1999 - Am Soc Nephrol Abstract. Polyomavirus (PV) exceptionally causes a morphologically manifest
renal allograft infection. Five such cases were encountered in this study, and
were followed between 40 and 330 d during persistent PV renal allograft ... Cited by 284 - Related articles - BL Direct - All 6 versions
PM Chesters, J Heritage, DJ McCance - The Journal of infectious diseases, 1983 - jstor.org Page 1. THE JOURNAL OF INFECTIOUS DISEASES * VOL. 147, NO. 4 * APRIL 1983 @ 1983
by The University of Chicago. All rights reserved. 0022-1899/83/4704-0010$00.88 ... Cited by 280 - Related articles - All 3 versions
RH Rubin, JS Wolfson, AB Cosimi, NE Tolkoff … - The American Journal of Medicine, 1981 - Elsevier The major types of infection observed can be categorized according to the time
period post-transplant in which they occur: postsurgical bacterial infection in
the first month after transplantation; opportunistic infection, with ... Cited by 262 - Related articles - All 4 versions
H Hirsch, D Brennan, C Drachenberg… - Transplantation, 2005 - Springer Abstract P olyomavirus-associated nephropathy (PVAN) is an emerging disease in
renal transplant patients with variable prevalence of 1-10% and graft loss up to
80%. BK virus (BKV) is the primary etiologic agent, but JC virus (JCV) and ... Cited by 257 - Related articles - BL Direct - All 7 versions
I Binet, V Nickeleit, HH Hirsch, O Prince, P … - Transplantation, 1999 - journals.lww.com Skip Navigation Links Home > March 27, 1999 - Volume 67 - Issue 6 > POLYOMAVIRUS
DISEASE UNDER NEW IMMUNOSUPPRESSIVE DRUGS: A Ca... ... 1 Division of
Nephrology, Department of Medicine. ... 2 Address correspondence to: ... Cited by 251 - Related articles - BL Direct - All 3 versions
HH Hirsch, J Steiger - The Lancet Infectious Diseases, 2003 - Elsevier Polyomavirus hominis 1, better known as BK virus (BKV), infects up to 90% of the
general population. However, significant clinical manifestations are rare and
limited to individuals with impaired immune functions. BKV has been ... Cited by 252 - Related articles - All 7 versions
- ►asnjournals.org E Ramos, CB Drachenberg, JC Papadimitriou, … - Journal of the American Society of Nephrology, 2002 - Am Soc Nephrol ABSTRACT. Polyoma virus (PV) can cause interstitial nephritis and lead to graft
failure in renal transplant recipients. The clinical course of patients with
polyoma virus nephritis (PVN) is not well understood, partially due to its ... Cited by 214 - Related articles - BL Direct - All 4 versions
