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Scholar Results 1 - 10 of about 18 citing NISHINO: Lupus erythematosus-like syndrome with selective complete deficiency of C1q. (0.26 sec) 

Hereditary C1q deficiency and systemic lupus erythematosus


P Bowness, KA Davies, PJ Norsworthy, P … - Qjm, 1994 - Oxford Univ Press
Page 1. QJMed 1994; 87:455-464 Original papers QJM Hereditary C1q deficiency
and systemic lupus erythematosus P. BOWNESS 1 *, KA ...
Cited by 122 - Related articles - BL Direct - All 3 versions

Complement and systemic lupus erythematosus


M Walport - Arthritis Res, 2002 - biomedcentral.com
Complement is implicated in the pathogenesis of systemic lupus erythematosus
(SLE) in several ways and may act as both friend and foe. Homozygous deficiency
of any of the proteins of the classical pathway is causally associated with ...
Cited by 75 - Related articles - Cached - All 7 versions

Lupus diseases associated with hereditary and acquired deficiencies of complement


V Agnello - Springer seminars in immunopathology, 1986 - Springer
The etiologies of Systemic lupus erythematosus (SLE) and other lupus and lupus-
like diseases are unknown, however evidence from both human and animal studies
suggests that multiple factors including genetic, endocrine, and ...
Cited by 47 - Related articles - All 3 versions

Systemic lupus erythematosus and complement deficiency: clues to a novel role for the …


JS Navratil, LC Korb, JM Ahearn - Immunopharmacology, 1999 - Elsevier
Complete deficiency of C1q, the first component of the classical pathway of
complement activation, is almost invariably associated with the development of
systemic lupus erythematosus. Understanding why complement deficiency ...
Cited by 34 - Related articles - All 4 versions

Mannose binding lectin: genetics and autoimmune disease


A Tsutsumi, R Takahashi, T Sumida - Autoimmunity Reviews, 2005 - Elsevier
Mannose binding lectin (MBL) is a serum protein with structure and functions
similar to those of complement factor C1q, and is a key molecule in innate
immunity. Interestingly, absence or extremely low concentration of serum ...
Cited by 30 - Related articles - All 2 versions

Genetic deficiencies of the complement system and association with disease-early …


K Kolble, KBM Reid - International Reviews of Immunology, 1993 - informahealthcare.com
Intern. Rev. Immunol. Vol. 10, 1993, pp. 17-36 Reprints available directly from
the publisher Photocopying permitted by license only O1993 Harwood Academic
Publishers GmbH Printed in the United States of America
Cited by 27 - Related articles - BL Direct - All 4 versions

SLE like syndrome and functional deficiency of C1q in members of a large family.

- nih.gov [PDF] 
AJ Hannema, JC Kluin-Nelemans, CE Hack, … - Clinical and Experimental Immunology, 1984 - pubmedcentral.nih.gov
Two sisters and a brother from one family are described whose sera were
deficient in haemolytic complement function. This defect was restored by
addition ofpurified C I q. In their sera, Clq like material was found, ...
Cited by 26 - Related articles - All 3 versions

A homozygous point mutation results in a stop codon in the C1q B-chain of a C1q-deficient …


RA McAdam, D Goundis, KBM Reid - Immunogenetics, 1988 - Springer
Page 1. Immunogenetics 27: 259-264, 1988 ll l llliO- genetics © Springer-Verlag
1988 A homozygous point mutation results in a stop codon in the ...
Cited by 27 - Related articles - All 3 versions

Role of C1q and Clq Receptors in the Pathogenesis of Systemic Lupus Erythematosus


B Ghebrehiweta, EIE Peerschke - Complement in Autoimmunity, 2003 - books.google.com
Tsokos GC (ed): Complement in Autoimmunity. Curr Dir Autoimmun. Basel, Karger,
2004, vol 7, pp 87-97 Role of Clq and Clq Receptors in the Pathogenesis of
Systemic Lupus Erythematosus Berhane Ghebrehiweta, Ellinor IE Peerschke0 ...
Cited by 20 - Related articles - BL Direct - All 5 versions

Complement receptors

- unair.ac.id [PDF] 
JM Ahearn, AM Rosengard - … : immunological mechanisms and prospects for new …, 1999 - books.google.com
12 Complement receptors JM AHEARN and AM ROSENGARD Complement receptor type 1
The primary function of complement receptor (CR) type 1 (CR1, CD35) is
phagocytosis and clearance of immune complexes, which is mediated by its ...
Cited by 19 - Related articles - All 2 versions


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