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Scholar Results 1 - 10 of about 48 citing Theos: Pathophysiology of neurofibromatosis type 1. (0.09 sec) 

Oncolytic HSV and erlotinib inhibit tumor growth and angiogenesis in a novel malignant …


YY Mahller, SS Vaikunth, MA Currier, SJ … - Molecular Therapy, 2007 - nature.com
Malignant peripheral nerve sheath tumors (MPNSTs), driven in part by hyperactive
Ras and epidermal growth factor receptor (EGFR) signaling, are often incurable.
Testing of therapeutics for MPNST has been hampered by lack of adequate ...
Cited by 22 - Related articles - All 2 versions

Malignant peripheral nerve sheath tumor: molecular pathogenesis and current management …


SR Grobmyer, JD Reith, A Shahlaee, CH … - Seminars in Surgical Oncology - interscience.wiley.com
Malignant Peripheral Nerve Sheath Tumors (MPNSTs) are rare tumors that often
occur in patients with neurofibromatosis 1. Surgical resection represents the
mainstay of treatment. Radiation and chemotherapy have a role in selected ...
Cited by 9 - Related articles - BL Direct - All 2 versions

Analysis of mitochondrial DNA in discordant monozygotic twins with neurofibromatosis type …


AK Detjen, S Tinschert, D Kaufmann, B … - Twin Research and Human Genetics, 2007 - Aus Acad Press
Neurofibromatosis type 1 (NF1) is the most frequent neurocutaneous disorder with
autosomal dominant inheritance. Phenotype variability is high ranging from
merely several café-au-lait spots to malignant periph- eral nerve sheath ...
Cited by 8 - Related articles - BL Direct - All 2 versions

Neurofibromatosis type I: genetics and clinical manifestations


A Savar, DM Cestari - Seminars in Ophthalmology, 2007 - informahealthcare.com
Neurofibromatosis type I (NF-1) was first described by Friedrich Daniel von
Recklinghausen in 1882. It is a common genetic disease with multisystem
involvement and a number of ophthalmologic findings that can involve the ...
Cited by 6 - Related articles - BL Direct - All 4 versions

Leukemia-associated NF1 inactivation in patients with pediatric T-ALL and AML lacking …

- hematologylibrary.org
BV Balgobind, P Van Vlierberghe, AMW van … - Blood, 2008 - bloodjournal.hematologylibrary.org
Neurofibromatosis type 1 (NF1) is an autosomal dominant genetic disorder caused
by mutations in the NF1 gene. Patients with NF1 have a higher risk to develop
juvenile myelomonocytic leukemia (JMML) with a possible progression toward ...
Cited by 6 - Related articles - BL Direct - All 2 versions

Magnetic resonance imaging of soft-tissue tumors: determinate and indeterminate lesions

- tums.ac.ir [PDF] 
DF Papp, AJ Khanna, EF McCarthy, JA Carrino … - The Journal of Bone and Joint Surgery, 2007 - JBJS
Disclosure: The authors did not receive any outside funding or grants in support
of their research for or preparation of this work. Neither they nor a member of
their immediate families received payments or other benefits or a ...
Cited by 7 - Related articles - BL Direct - All 3 versions

Genodermatoses with malignant potential

- prsjournal.com
JD Holman, JA Dyer - Current opinion in pediatrics, 2007 - journals.lww.com
Purpose of review: The delineation of syndromes carrying a predisposition to
malignancy has led to great insights into the molecular biology of malignancy.
Many such syndromes have cutaneous findings which can precede the ...
Cited by 6 - Related articles - BL Direct - All 4 versions

McCune-Albright syndrome


CE Dumitrescu, MT Collins - Orphanet Journal of Rare Diseases, 2008 - ojrd.com
McCune-Albright syndrome (MAS) is classically defined by the clinical triad of
fibrous dysplasia of bone (FD), café-au-lait skin spots, and precocious puberty
(PP). It is a rare disease with estimated prevalence between 1/100,000 and ...
Cited by 6 - Related articles - Cached - All 14 versions

p53 signaling in response to increased DNA damage sensitizes AML1-ETO cells to stress- …

- hematologylibrary.org
O Krejci, M Wunderlich, H Geiger, FS Chou, … - Blood, 2008 - bloodjournal.hematologylibrary.org
Chromosomal translocation (8;21) is present in 10% to 15% of patients with acute
myeloid leukemia. Expression of the AML1-ETO (AE) fusion protein alone is not
sufficient to induce leukemia, but the nature of the additional genetic ...
Cited by 6 - Related articles - BL Direct - All 6 versions

[PDF] Leukemia associated NF1 inactivation in pediatric T-ALL and AML patients lacking evidence …


BV Balgobind, P Van Vlierberghe, AMW van … - Molecular-genetic insights in pediatric T-cell acute …, 2008 - publishing.eur.nl
Chapter 8 Leukemia associated NF1 inactivation in pediatric T-ALL and AML
patients lacking evidence for neurofibromatosis Brian V. Balgobind1*, Pieter Van
Vlierberghe1*, Ans MW van den Ouweland2, H. Berna Beverloo2, Joan NR ...
Cited by 4 - Related articles - View as HTML


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