- ►diabetesjournals.org M Furuhashi, N Ura, T Nakata, K Shimamoto - Diabetes Care, 2003 - Am Diabetes Assoc RESULTS—Diabetes was identified in 12 (20%) of the 61 CD36-deficient patients.
Fasting glucose, HbA 1c , and total cholesterol levels in the diabetic
CD36-deficient patients were significantly higher than in the control ... Cited by 19 - Related articles - BL Direct - All 5 versions
F Lepretre, KJ Linton, C Lacquemant, V Vatin, … - Diabetes and Metabolism, 2004 - Elsevier We detected five mutations, P191P and N247S were only found each in one family
and did not segregate with diabetes, the three others (A/C-178 in the promoter,
A/G-10 in intron 3 and (GGGTTGAGA) insertion in intron 13) being equally ... Cited by 15 - Related articles - All 3 versions
M Furuhashi, N Ura, T Nakata, T Tanaka, K … - Diabetic Medicine, 2004 - interscience.wiley.com It is also possible that your web browser is not configured or not able to
display style sheets. In this case, although the visual presentation will be
degraded, the site should continue to be functional. We recommend using the ... Cited by 7 - Related articles - BL Direct - All 4 versions
- ►diabetesjournals.org [PDF] T Kuwasako, K Hirano, N Sakai, M Ishigami, … - Diabetes Care, 2003 - Am Diabetes Assoc Our objective was to evaluate the di- agnostic proficiency of the World Health
Organization (WHO) and the National Cholesterol Education Pro- gram (NCEP)-III
definitions (1,2) for the metabolic syndrome in a Mexican nation- wide, ... Cited by 13 - Related articles - BL Direct - All 5 versions
E Corpeleijn, CJ van der Kallen, M … - Diabetic Medicine, 2006 - interscience.wiley.com It is also possible that your web browser is not configured or not able to
display style sheets. In this case, although the visual presentation will be
degraded, the site should continue to be functional. We recommend using the ... Cited by 15 - Related articles - BL Direct - All 5 versions
F Lepretre, F Vasseur, M Vaxillaire, PE Scherer … - Human Mutation, 2004 - interscience.wiley.com Mutations in CD36 / fatty acid translocase (FAT) gene are responsible for
insulin resistance in the rat but contribution to human Type 2 diabetes is
unknown. A nominal evidence for linkage of familial T2D at the CD36 locus ... Cited by 25 - Related articles - All 2 versions
S Kajihara, A Hisatomi, Y Ogawa, T Yasutake, … - Clinica Chimica Acta, 2001 - Elsevier Background: CD36 deficiency is reportedly an underlying factor about insulin
resistance, defective fatty acid metabolism and hypertriglyceridemia in
spontaneously hypertensive rat (SHR), and may be involved in the ... Cited by 6 - Related articles - All 4 versions
X Ma, S Bacci, W Mlynarski, L Gottardo, T … - Human Molecular Genetics, 2004 - Oxford Univ Press Xiaowei Ma 1,2 , Simonetta Bacci 3 , Wojciech Mlynarski 1,2 , Lucia Gottardo 4 ,
Teresa Soccio 1,2 , Claudia ... Menzaghi 3 , Elisabetta Iori 4 , Robert A.
Lager 2,5* , Adhir R. Shroff 2,5 , Ernest V. Gervino 2,5 , Richard W. Cited by 44 - Related articles - BL Direct - All 8 versions
- ►jlr.org T Tanaka, T Nakata, T Oka, T Ogawa, F … - The Journal of Lipid Research, 2001 - ASBMB Because of the importance of long-chain fatty acids (LCFAs) as a myocardial
energy substrate, myocardial LCFA metabolism has been of particular interest for
the understanding of cardiac pathophysiology. Recently, by using ... Cited by 46 - Related articles - BL Direct - All 4 versions
H Yanai, H Chiba, H Fujiwara, M Morimoto, Y … - Thrombosis and haemostasis, 2001 - cat.inist.fr Previous in vitro studies have shown that CD 36 participates in cellular fatty
acid(FA) uptake. In vivo evidence for a physiologic role of CD 36 in this
process is poor and mostly obtained in animals. To examine the metabolic ... Cited by 12 - Related articles - BL Direct - All 3 versions
