Web Images Videos Maps News Shopping Gmail more »
Sign in
Scholar Home  
  Advanced Scholar Search
Scholar Preferences
Scholar Results 1 - 10 of about 101 related to Perseghin: Contribution of abnormal insulin secretion and insulin resistance to the pathogenesis.... (0.44 sec) 

Contribution of abnormal insulin secretion and insulin resistance to the pathogenesis of …


G Perseghin, A Caumo, C Arcelloni, S Benedini … - Diabetes care, 2003 - Am Diabetes Assoc
RESULTS—MyD had reduced lean body mass, but peripheral insulin sensitivity was
not different to that of control subjects in contrast to OFF, which showed
insulin resistance. Insulin secretion, obtained by deconvolution of OGTT ...
Cited by 14 - Related articles - BL Direct - All 5 versions

Postabsorptive and insulin-stimulated energy and protein metabolism in patients with …

- cnfindpark.cn
G Perseghin, M Comola, P Scifo, S Benedini, F … - American Journal of Clinical Nutrition, 2004 - Am Soc Nutrition
Results: Patients with DM1 had less lean body mass, greater fat mass, and
greater intramyocellular lipid contents than did healthy control subjects.
Energy expenditure and glucose and lipid metabolism did not differ ...
Cited by 8 - Related articles - BL Direct - All 6 versions

DM1 CTG expansions affect insulin receptor isoforms expression in various tissues of …


C Guiraud-Dogan, A Huguet, M Gomes-Pereira … - BBA-Molecular Basis of Disease, 2007 - Elsevier
Myotonic dystrophy (DM1) is a dominant autosomal multisystemic disorder caused
by the expansion of an unstable CTG trinucleotide repeat in the 3′
untranslated region of the DMPK gene. Nuclear accumulation of the enlarged ...
Cited by 4 - Related articles - All 3 versions

Hyperproinsulinaemia in patients with myotonic dystrophy


AJ Krentz, PM Clark, L Cox, AC Williams, M … - Diabetologia, 1992 - Springer
38 _+ 4 pmol/1, p = 0.09) were not significantly different be- tween the groups.
Fasting concentrations of proinsulin (10.3 + 2.9vs 1.6 +_ 0.3 pmol/1,p < 0.01),
and 32-33 split proin- sulin (7.8 + 2.5 vs 2.9 + 0.4 pmol/1,p < 0.05) were ...
Cited by 9 - Related articles - BL Direct - All 3 versions

[CITATION] Regulation of expression of 11beta-hydroxysteroid dehydrogenase dehydrogenase type 1 in …


JW Tomlinson, J Moore, MD Cooper… - Endocrinology, 2001
Cited by 2 - Related articles

[CITATION] Subcutaneous adipose 11beta-hydroxysteroid dehydrogenase type 1 and messenger …


RS Lindsay, DJ Wake, S Nair… - J Clin Endocrinol Metab, 2003
Cited by 2 - Related articles

[CITATION] In: Engel AG, Banker BQ, eds. Myology


PS Harper, 1986 - New York: McGraw-Hill
Cited by 2 - Related articles

Dubious assumptions underlying the adjustment of metabolic rates for changes in fat-free …

- bodieslikeours.org [PDF] 
FF Mass - Journal of Clinical Endocrinology & Metabolism - Endocrine Soc
Received February 28, 2003. Address correspondence to: Dympna Gallagher, Ed.D.,
St. Luke's Roosevelt Hospital, Obesity Research Cen- ter, Columbia University,
1090 Amsterdam Avenue, 14th Floor, New York, New York 10025. E-mail: dg108@ ...
Cited by 3 - Related articles - BL Direct - All 6 versions

Relationship between insulin secretion and insulin resistance in nondiabetic subjects with …


T Goto, T Sato, T Hinata, S Suga, T Onuma, … - Nō to shinkei= Brain and nerve, 1995 - ncbi.nlm.nih.gov
Hyperinsulinemia in myotonic dystrophy (MyD) is generally considered as a result
of insulin resistance. However, as a consequence of abnormality in systemic
membrane fluidity in this disease, abnormal insulin clearance or intrinsic ...
Cited by 3 - Related articles - BL Direct

Testosterone and diurnal rhythmicity of leptin, TNF-alpha and TNF-II receptor in insulin- …


A Johansson, B Ahren, H Forsberg, T Olsson - International journal of obesity and related metabolic …, 2002 - ncbi.nlm.nih.gov
1: Int J Obes Relat Metab Disord. 2002 Oct;26(10):1386-92. Testosterone
and diurnal rhythmicity of leptin, TNF-alpha and TNF-II ...
Cited by 13 - Related articles - BL Direct - All 2 versions


Result Page: 

1

2

3

4

5

6

7

8

9

10

Next


 


Go to Google Home - About Google - About Google Scholar

©2009 Google