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Scholar Results 1 - 10 of about 101 related to Stirban: Benfotiamine prevents macro-and microvascular endothelial dysfunction and oxidative.... (0.10 sec) 

Benfotiamine prevents macro-and microvascular endothelial dysfunction and oxidative stress …

- diabetesjournals.org
A Stirban, M Negrean, B Stratmann, T … - Diabetes Care, 2006 - Am Diabetes Assoc
RESEARCH DESIGN AND METHODS— Thirteen people with type 2 diabetes were given a
heat-processed test meal with a high AGE content (HAGE; 15.100 AGE kU, 580 kcal,
54 g protein, 17 g lipids, and 48 g carbohydrates) before and after a 3-day ...
Cited by 87 - Related articles - BL Direct - All 5 versions

Single oral challenge by advanced glycation end products acutely impairs endothelial …


J Uribarri, A Stirban, D Sander, W Cai, M … - Diabetes Care, 2007 - Am Diabetes Assoc
RESULTS—The diabetic patients had higher baseline levels of serum AGEs (P =
0.020), PAI-1 (NS), and VCAM-1 (P = 0.033) and lower baseline values of FMD
compared with nondiabetic subjects (P = 0.032). Ninety minutes after a ...
Cited by 33 - Related articles - BL Direct - All 4 versions

Benfotiamine in the treatment of diabetic polyneuropathy--a three-week randomized, …


E Haupt, H Ledermann, W Köpcke - International journal of clinical pharmacology and …, 2005 - ncbi.nlm.nih.gov
OBJECTIVE: The aim of the study was to evaluate the efficacy of benfotiamine
administered over three weeks (allithiamine; a lipid-soluble vitamin B1 prodrug
with high bioavailability) to patients with diabetic polyneuropathy in a ...
Cited by 47 - Related articles - All 2 versions

Effects of low-and high-advanced glycation endproduct meals on macro-and microvascular …

- cnfindpark.cn
M Negrean, A Stirban, B Stratmann, T … - American Journal of Clinical Nutrition, 2007 - ajcn.cnfindpark.cn
Design: Twenty inpatients with T2DM [ (±SEM) age: 55.4 ± 2.2 y; glycated
hemoglobin: 8.8 ± 0.5%] were investigated. In a randomized crossover design,
the effects of a low-AGE (LAGE) and high-AGE (HAGE) meal on macrovascular ...
Cited by 50 - Related articles - BL Direct - All 7 versions

Benfotiamine accelerates the healing of ischaemic diabetic limbs in mice through protein …


S Gadau, C Emanueli, S Van Linthout, G … - Diabetologia, 2006 - Springer
Page 1. Diabetologia (2006) 49: 405–420 DOI 10.1007/s00125-005-0103-5 ARTICLE S.
Gadau . C. Emanueli . S. Van Linthout . G. Graiani . M. Todaro . M. Meloni . ...
Cited by 47 - Related articles - BL Direct - All 6 versions

Advanced glycoxidation end products in commonly consumed foods


T Goldberg, W Cai, M Peppa, V Dardaine, BS … - Journal of the American Dietetic Association, 2004 - Elsevier
Foods of the fat group showed the highest amount of AGE content with a mean of
100±19 kU/g. High values were also observed for the meat and meat-substitute
group, 43±7 kU/g. The carbohydrate group contained the lowest values of ...
Cited by 120 - Related articles - All 12 versions

Benfotiamine alleviates diabetes-induced cerebral oxidative damage independent of …

- uwyo.edu [PDF] 
S Wu, J Ren - Neuroscience letters, 2006 - Elsevier
Diabetes mellitus leads to thiamine deficiency and multiple organ damage
including diabetic neuropathy. This study was designed to examine the effect of
benfotiamine, a lipophilic derivative of thiamine, on streptozotocin (STZ)- ...
Cited by 23 - Related articles - All 4 versions

Diet-derived advanced glycation end products are major contributors to the body's AGE pool …


J Uribarri, W Cai, O Sandu, M Peppa, T … - ANNALS-NEW YORK ACADEMY OF SCIENCES, 2005 - interscience.wiley.com
Abstract: Advanced glycation end products (AGEs) are a heterogeneous group of
compounds that form continuously in the body. Their rate of endogenous formation
is markedly increased in diabetes mellitus, a condition in which AGEs play ...
Cited by 60 - Related articles - BL Direct - All 5 versions

Adverse effects of dietary glycotoxins on wound healing in genetically diabetic mice

- diabetesjournals.org
M Peppa, H Brem, P Ehrlich, JG Zhang, W Cai, … - Diabetes, 2003 - Am Diabetes Assoc
Advanced glycoxidation end products (AGEs) are implicated in delayed diabetic
wound healing. To test the role of diet-derived AGE on the rate of wound
healing, we placed female db/db (+/+) (n = 55, 12 weeks old) and ...
Cited by 50 - Related articles - BL Direct - All 4 versions

Fetal or neonatal low-glycotoxin environment prevents autoimmune diabetes in NOD mice

- diabetesjournals.org
M Peppa, C He, M Hattori, R McEvoy, F Zheng … - Diabetes, 2003 - Am Diabetes Assoc
Advanced glycation end products (AGEs) are implicated in β-cell oxidant stress.
Diet-derived AGE (dAGE) are shown to contribute to end-organ toxicity attributed
to diabetes. To assess the role of dAGE on type 1 diabetes, NOD mice were ...
Cited by 34 - Related articles - BL Direct - All 5 versions


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