LA Suzuki, M Poot, RG Gerrity, KE Bornfeldt - Diabetes, 2001 - Am Diabetes Assoc In combination with other factors, hyperglycemia may cause the accelerated progression of atherosclerosis
in people with diabetes. Arterial smooth muscle cell (SMC) proliferation and accumulation contribute
to formation of advanced atherosclerotic lesions. Therefore, we investigated the effects of ... Cited by 119 - Related articles - BL Direct - All 5 versions
G Levels - DIABETES, 2001 - Am Diabetes Assoc In combination with other factors, hyperglycemia may cause the accelerated progression of atherosclerosis
in people with diabetes. Arterial smooth muscle cell (SMC) proliferation and accumulation contribute
to formation of advanced atherosclerotic lesions. Therefore, we in- vestigated the effects ... Related articles
H Fukumoto, Z Naito, G Asano, T Aramaki - Journal of atherosclerosis …, 1998 - ncbi.nlm.nih.gov Immunohistochemical and morphometrical studies were performed to elucidate the specificity
of atherosclerosis in the descending branch (the segments 5 and 6) of the left coronary artery
associated with acute myocardial infarction (AMI) in the anterior wall of the heart and non- ... Cited by 57 - Related articles
R Assert, G Scherk, A Bumbure, V Pirags, H Schatz, … - Diabetologia, 2001 - Springer Aims/hypothesis. Postprandial hyperglycaemia car- ries an increased risk of macrovascular disease
even without Type II (non-insulin-dependent) diabetes mellitus. Chronic hyperglycaemia activates
protein kinase C (PKC) in vitro and in vivo but it is not known whether PKC is regulated by ... Cited by 64 - Related articles - BL Direct - All 4 versions
A Ceriello, D Giugliano, A Quatraro, E Marchi, M … - Diabetologia, 1990 - Springer The key event in the coagulation of blood is the conver- sion of prothrombin to thrombin. Once
generated, this enzyme converts fibrinogen to fibrin with the release of fibrinopeptide A and B
[1]. Alternatively, throm- bin may be inhibited by antithrombinIII (ATIII), the most relevant ... Cited by 43 - Related articles - All 2 versions
AI Vinik, T Erbas, TS Park, R Nolan, GL Pittenger - Diabetes Care, 2001 - Am Diabetes Assoc Insulin resistance is a uniform finding in type 2 diabetes, as are abnormalities in the microvascular
and macrovascular circulations. These complications are associated with dysfunction of platelets
and the neurovascular unit. Platelets are essential for hemostasis, and knowledge of their ... Cited by 254 - Related articles - BL Direct - All 5 versions
[CITATION] Is insulin resistance or diabetes mellitus associated with stroke
H Adachi, Y Hirai, M Tsuruta, Y Fujiura, T Imaizuml - Diabetes Res Clin Pract, 2001 Cited by 15 - Related articles
- ►nih.gov [PDF] A Sniderman, D Thomas, D Marpole, B … - Journal of Clinical …, 1978 - pubmedcentral.nih.gov ABS TRA C T The mechanism(s) by whichcholes- terol returns to the splanchnic bed from peripheral
tissues are not well understood. To study this phe- nomenon in fasting man, lipoproteins were
isolated from plasma obtained from hepatic vein and aorta. Cholesterol content of each ... Cited by 47 - Related articles - All 6 versions
- ►diabetesjournals.org CE Hafer-Macko, FM Ivey, KA Gyure, JD Sorkin, RF … - Diabetes, 2002 - Am Diabetes Assoc Human diabetic neuropathy is multifactorial in etiology, with ischemia as a final common
pathology. Although impaired vascular endothelial cell function in diabetic microvascular injury
is established, the role of thrombomodulin (TM)-dependent protein C antithrombotic ... Cited by 39 - Related articles - BL Direct - All 5 versions
- ►physiology.org Y Li, V Woo, R Bose - American Journal of Physiology- Heart and …, 2001 - Am Physiological Soc We sought to determine the mechanisms for hyperactivity and abnormal platelet Ca 2+ homeostasis
in diabetes. The glycosylated Hb (HbA 1c ) level was used as an index of glycemic control. Human
platelets were loaded with Ca- green-fura red, and cytosolic Ca 2+ ([Ca 2+ ] i ) and ... Cited by 98 - Related articles - BL Direct - All 4 versions
