- ►diabetesjournals.org F Tremblay, C Lavigne, H Jacques, A Marette - Diabetes, 2001 - Am Diabetes Assoc The cellular mechanism by which high-fat feeding induces skeletal muscle insulin resistance was
investigated in the present study. Insulin-stimulated glucose transport was impaired (∼40–60%)
in muscles of high fat–fed rats. ... Cited by 103 - Related articles - BL Direct - All 5 versions
[CITATION] Chapitre 1 Le défaut de translocation de GLUT4 dans le muscle squelettique de …
PA Hansen, DH Han, BA Marshall, LA Nolte, MM … - Journal of Biological …, 1998 - ASBMB A high fat diet causes resistance of skeletal muscle glucose transport to insulin and contractions.
We tested the hypothesis that fat feeding causes a change in plasma membrane composition that
interferes with functioning of ... Cited by 105 - Related articles - BL Direct - All 6 versions
- ►endojournals.org Y Kanoh, G Bandyopadhyay, MP Sajan, ML Standaert, … - Endocrinology, 2001 - Endocrine Soc Atypical protein kinases C (PKCs), and , and protein kinase B (PKB) are thought to function
downstream of phosphatidylinositol 3-kinase (PI 3-kinase) and regulate glucose transport during
insulin action in skeletal muscle ... Cited by 42 - Related articles - BL Direct - All 4 versions
GJ Etgen, KM Valasek, CL Broderick, AR Miller - Journal of Biological Chemistry, 1999 - ASBMB An in vivo adenoviral gene delivery system was utilized to assess the effect of overexpressing
protein kinase C (PKC)-ζ on rat skeletal muscle glucose transport activity. Female lean Zucker rats
were injected with adenoviral/human ... Cited by 61 - Related articles - All 5 versions
- ►endojournals.org Y Kanoh, MP Sajan, G Bandyopadhyay, A Miura, ML … - Endocrinology, 2003 - Endocrine Soc Insulin-stimulated glucose transport in skeletal muscle is thought to be effected at least partly
through atypical protein kinase C isoforms (aPKCs) operating downstream of phosphatidylinositol (PI)
3-kinase and 3-phosphoinositide- ... Cited by 30 - Related articles - BL Direct - All 3 versions
- ►csun.edu [PDF] MK Singh, AD Krisan, AM Crain, DE Collins, BB … - Metabolism, 2003 - Elsevier The aim of this investigation was to evaluate if leptin treatment enhances insulin-stimulated
glucose transport in normal (experimental group [EXP]-1) and insulin-resistant skeletal muscle
(EXP-2) by altering components of ... Cited by 25 - Related articles - All 9 versions
- ►diabetesjournals.org F Tremblay, C Lavigne, H Jacques, A Marette - Diabetes, 2003 - Am Diabetes Assoc Diet-induced obesity is known to cause peripheral insulin resistance in rodents. We have recently
found that feeding cod protein to high-fat-fed rats prevents the development of insulin resistance
in skeletal muscle. In the present ... Cited by 34 - Related articles - BL Direct - All 6 versions
[CITATION] Chapitre 2 La protéine de morue rétablit l'activation par l'insuline de la voie PI 3- …
- ►physiology.org AE Halseth, DP Bracy, DH … - American Journal of …, 2000 - Am Physiological Soc Rats fed a high-fat diet display blunted insulin-stimulated skeletal muscle glucose uptake. It is
not clear whether this is due solely to a defect in glucose transport, or if glucose delivery and
phosphorylation are also impaired. ... Cited by 38 - Related articles - BL Direct - All 3 versions
