K Moens, V Berger, JM Ahn, C Van … - Diabetes, 2002 - Am Diabetes Assoc Glucagon is a potent stimulator of insulin release in the presence of a
permissive glucose concentration, activating β-cells in vitro via both
glucagon- and glucagon-like peptide-1 (GLP-1)-receptors. It is still ... Cited by 20 - Related articles - BL Direct - All 5 versions
P Huypens, Z Ling, D Pipeleers, F Schuit - Diabetologia, 2000 - Springer Previous studies on rat beta cells in vitro have sug- gested that insulin
release is synergistically regulated by signalling molecules derived from
glucose metabo- lism on the one hand and adenylate cyclase stimula- tion by ... Cited by 58 - Related articles - BL Direct - All 4 versions
S Dalle, C Longuet, S Costes, C Broca, O … - Journal of Biological Chemistry, 2004 - ASBMB Page 1. 1 Glucagon promotes CREB phosphorylation via activation of ERK1/2
(extracellular signal-related kinase 1/2) in MIN6 cell line ... Cited by 2 - Related articles - All 2 versions
- ►diabetesjournals.org S Dalle, G Fontés, AD Lajoix, L LeBrigand, R … - Diabetes, 2002 - Am Diabetes Assoc Miniglucagon, the COOH-terminal (19-29) fragment processed from glucagon, is a
potent and efficient inhibitor of insulin secretion from the MIN 6 ß-cell line.
Using the rat isolated-perfused pancreas, we investigated the inhibitory ... Cited by 17 - Related articles - BL Direct - All 4 versions
K Kawai, C Yokota, S Ohashi, Y Watanabe, K … - Diabetologia, 1995 - Springer Summary Since glucagon-like peptide-1 (7-36) amide (7-37) (GLP-1) has been found
to be a potent insu- linotropic hormone, it has been postulated that gluca- gon
stimulates insulin secretion from islet beta cells through the GLP-1 ... Cited by 30 - Related articles - BL Direct - All 3 versions
- ►jbc.org S Dalle, P Smith, P Blache, D Le-Nguyen, L Le … - Journal of Biological Chemistry, 1999 - ASBMB Using the MIN6 B-cell line, we investigated the hypothesis that miniglucagon,
the C-terminal (19-29) fragment processed from glucagon and present in
pancreatic A cells, modulates insulin release, and we analyzed its cellular ... Cited by 29 - Related articles - BL Direct - All 5 versions
- ►shouxi.net K Hussain, J Bryan, HT Christesen, K … - Diabetes, 2005 - Am Diabetes Assoc The mechanisms involved in the release of glucagon in response to hypoglycemia
are unclear. Proposed mechanisms include the activation of the autonomic nervous
system via glucose-sensing neurons in the central nervous system, via the ... Cited by 22 - Related articles - BL Direct - All 7 versions
K Hussain - Horm Res, 2008 - content.karger.com Hyperinsulinaemic hypoglycaemia is a cause of persistent hypoglycaemia in the
neonatal and infancy periods. Prompt recognition and management of patients with
hyperinsulinaemic hypoglycaemia are essential, if brain damage and ... Cited by 12 - Related articles - BL Direct - All 5 versions
[CITATION] &Rao A (1996). Recombinant NFAT1 (NFATp) is regulated by calcineurin in T cells and …
O Fafoula, H Alkhayyat, K Hussain - Archives of disease in childhood. Fetal and neonatal …, 2006 - ncbi.nlm.nih.gov 1: Arch Dis Child Fetal Neonatal Ed. 2006 Nov;91(6):F467. Prolonged hyperinsulinaemic
hypoglycaemia in newborns with intrauterine growth retardation. ... Cited by 2 - Related articles - BL Direct - All 3 versions
