SM Hofmann, HJ Dong, Z Li, W Cai, J … - Diabetes, 2002 - Am Diabetes Assoc Advanced glycation end products (AGEs), known promoters of diabetic
complications, form abundantly in heated foods and are ingested in bioreactive
forms. To test whether dietary AGEs play a role in the progression of ... Cited by 115 - Related articles - BL Direct - All 5 versions
F Zheng, C He, W Cai, M Hattori, M Steffes, H … - Diabetes/Metabolism Reviews - interscience.wiley.com It is also possible that your web browser is not configured or not able to
display style sheets. In this case, although the visual presentation will be
degraded, the site should continue to be functional. We recommend using the ... Cited by 88 - Related articles - BL Direct - All 4 versions
RY Lin, ED Reis, AT Dore, M Lu, N Ghodsi, JT … - Atherosclerosis, 2002 - Elsevier Restenosis remains a major cause of morbidity and mortality after coronary
angioplasty. Injury-induced inflammation, thrombosis, smooth muscle cell (SMC)
proliferation, and neointimal formation contribute to restenosis. These ... Cited by 70 - Related articles - All 9 versions
- ►diabetesjournals.org M Peppa, C He, M Hattori, R McEvoy, F Zheng … - Diabetes, 2003 - Am Diabetes Assoc Advanced glycation end products (AGEs) are implicated in β-cell oxidant stress.
Diet-derived AGE (dAGE) are shown to contribute to end-organ toxicity attributed
to diabetes. To assess the role of dAGE on type 1 diabetes, NOD mice were ... Cited by 34 - Related articles - BL Direct - All 5 versions
- ►diabetesjournals.org M Peppa, H Brem, P Ehrlich, JG Zhang, W Cai, … - Diabetes, 2003 - Am Diabetes Assoc Advanced glycoxidation end products (AGEs) are implicated in delayed diabetic
wound healing. To test the role of diet-derived AGE on the rate of wound
healing, we placed female db/db (+/+) (n = 55, 12 weeks old) and ... Cited by 50 - Related articles - BL Direct - All 4 versions
- ►nih.gov H Vlassara, W Cai, J Crandall, T Goldberg, R … - Proceedings of the National Academy of Sciences of …, 2002 - National Acad Sciences Diet is a major environmental source of proinflammatory AGEs (heat-generated
advanced glycation end products); its impact in humans remains unclear. We
explored the effects of two equivalent diets, one regular (high AGE, H-AGE) ... Cited by 228 - Related articles - BL Direct - All 10 versions
- ►diabetesjournals.org O Sandu, K Song, W Cai, F Zheng, J Uribarri, … - Diabetes, 2005 - Am Diabetes Assoc Dietary advanced glycosylation end products (AGEs) have been linked to insulin
resistance in db/db (++) mice. To test whether dietary AGEs play a role in the
progression of insulin resistance in normal mice fed high-fat diets, normal ... Cited by 47 - Related articles - BL Direct - All 6 versions
RY Lin, RP Choudhury, W Cai, M Lu, JT Fallon … - Atherosclerosis, 2003 - Elsevier Hyperglycemia derived advanced glycation endproducts (AGE) have been implicated
in diabetic atherosclerosis (AS) but the role of exogenous (dietary) AGE in the
development of this serious complication is not known. This study evaluates ... Cited by 77 - Related articles - All 10 versions
- ►diabetesjournals.org [PDF] C He, J Sabol, T Mitsuhashi, H Vlassara - Diabetes, 1999 - Am Diabetes Assoc Evidence indicates that the metabolic turnover of food- derived reactive orally
absorbed advanced glycation end products (AGEs) or glycotoxins (GTs) is delayed,
pos- sibly contributing to the tissue damage induced by endogenous AGEs, ... Cited by 61 - Related articles - BL Direct - All 5 versions
