M Haluzik, KR Dietz, JK Kim, B Marcus- … - Diabetes, 2002 - Am Diabetes Assoc The virtually fatless A-ZIP/F-1 mouse is profoundly insulin resistant, diabetic,
and a good model for humans with severe generalized lipoatrophy. Like a number
of other mouse models of diabetes, the A-ZIP/F-1 mouse has elevated serum ... Cited by 33 - Related articles - BL Direct - All 4 versions
JN MacLeod, BH Shapiro - Laboratory animal science, 1988 - ncbi.nlm.nih.gov A re-usable mobile catheterization apparatus was developed that permits multiple
blood samples to be obtained from nonrestrained and unanesthetized mice. The
collection end of the catheter is located outside the cage allowing the ... Cited by 33 - Related articles - All 2 versions
- ►diabetesjournals.org [PDF] O Gavrilova, B Marcus-Samuels, ML Reitman - Diabetes, 2000 - Am Diabetes Assoc Stimulation of ß3-adrenergic receptors increases meta- bolic rate via lipolysis
in white adipose tissue (WAT) and thermogenesis in brown adipose tissue (BAT).
Other acute effects include decreased gastrointestinal motil- ity and food ... Cited by 34 - Related articles - BL Direct - All 4 versions
E Degerman, T Rahn-Lanstrom, L Stenson- … - Diabetes Mellitus: A Fundamental and Clinical Text, 2004 - books.google.com 374 Diabetes Mellitus: A Fundamental and Clinical Text explained by its ability
to lower cAMP and PKA (15-18). Fur- thermore, insulin-induced activation of
adipocyte PDE3B, me- diated by insulin-stimulated kinases [including ... Cited by 7 - Related articles
M Hasegawa, Y Tang, H Osawa, H Onuma, T … - Diabetes research and clinical practice, 2002 - Elsevier Phosphodiesterase (PDE) 3B, a major isoform of PDE in adipocytes, mediates the
antilipolytic action of insulin. PDE3B gene expression is generally reduced in
adipocytes of either monogenic or polygenic rodent models of obese, insulin- ... Cited by 7 - Related articles - All 9 versions
- ►nih.gov JK Kim, MD Michael, SF Previs, OD Peroni, F … - Journal of Clinical Investigation, 2000 - Am Soc Clin Investig Obesity and insulin resistance in skeletal muscle are two major factors in the
pathogenesis of type 2 diabetes. Mice with muscle-specific inactivation of the
insulin receptor gene (MIRKO) are normoglycemic but have increased fat ... Cited by 159 - Related articles - BL Direct - All 8 versions
