E Bernal-Mizrachi, W Wen, M Shornick, MA … - Diabetes, 2002 - Am Diabetes Assoc The purpose of the current study was to determine whether nuclear factor-κB
(NF-κB) activation is a component of the depolarization/Ca 2+ -dependent
signaling in β-cells. MIN6 cells were transfected with a plasmid ... Cited by 20 - Related articles - BL Direct - All 5 versions
RL Nolsøe, YH Hamid, F Pociot, S Paulsen, … - Genes and Immunity, 2006 - nature.com Type II diabetes is caused by a failure of the pancreatic -cells to compensate
for insulin resistance leading to hyperglycaemia. There is evidence for an
essential role of an increased -cell apoptosis in type II diabetes. High ... Cited by 7 - Related articles - BL Direct - All 3 versions
- ►shouxi.net S Norlin, U Ahlgren, H Edlund - Diabetes, 2005 - Am Diabetes Assoc Glucose-stimulated insulin secretion (GSIS) in pancreatic β-cells depends on
coordinated glucose uptake, oxidative metabolism, and Ca 2+ -triggered insulin
exocytosis. Impaired GSIS is a hallmark of type 2 diabetes. However, at ... Cited by 41 - Related articles - All 7 versions
LA Borg, DL Eizirik - Immunology letters, 1990 - ncbi.nlm.nih.gov 1: Immunol Lett. 1990 Dec;26(3):253-8. Short-term exposure of rat pancreatic islets
to human interleukin-1 beta increases cellular uptake of calcium. ... Cited by 12 - Related articles - All 3 versions
E Jaimovich, MA Carrasco - Biol. Res, 2002 - SciELO Chile Potassium depolarization of cultured muscle cells was employed to study cellular
responses linked to calcium signaling. Events occurring after depolarization
include i) A transient increase of the IP 3 mass (2-10s); ii) A slow ... Cited by 25 - Related articles - Cached - BL Direct - All 8 versions
[CITATION] The NEDD8 pathway is essential for SCFβ− TrCP-mediated ubiquitination and processing of …
S Helqvist, P Bouchelouche, HU Andersen, J … - Acta endocrinologica, 1989 - EFES The controlled flux of calcium across the cell membrane is intimately linked to
the release of insulin from pancreatic beta-cells, but the uncontrolled influx
of calcium is a common final denominator of cell death. Because interleukin ... Cited by 4 - Related articles - All 7 versions
- ►diabetesjournals.org K Maedler, DM Schumann, N Sauter, H … - Diabetes, 2006 - Am Diabetes Assoc High glucose concentrations have a dual effect on β-cell turnover, inducing
proliferation in the short-term and apoptosis in the long-term. Hyperglycemia
leads to β-cell production of interleuking (IL)-1β in human pancreatic ... Cited by 30 - Related articles - BL Direct - All 4 versions
N Welsh, T Nilsson, A Hallberg, P Arkhammar … - European Journal of Endocrinology, 1989 - EFES Isolated islets from adult rats or obese hyperglycemic (ob/ob) mice were
incubated with human recombinant interleukin 1β in order to study whether the
acute effects of the cytokine on islet insulin release are associated with ... Cited by 5 - Related articles - All 5 versions
TP Liu, PC Yu, I Liu… - Autonomic Neuroscience, 2002 - Elsevier Regulation of glucose metabolism by cholinergic nervous activation has been
demonstrated. In an attempt to evaluate the role of cholinergic receptor subtype
in this regulation of glucose metabolism, we employed cultured myoblast C 2 ... Cited by 7 - Related articles - All 6 versions
