M Muranyi, M Fujioka, QP He, A Han, G Yong, K … - Diabetes, 2003 - Am Diabetes Assoc It is well known that diabetes aggravates brain damage in experimental and clinical stroke
subjects. Diabetes accelerates maturation of neuronal damage, increases infarct volume, and
induces postischemic seizures. The mechanism by which diabetes increases ischemic ... Cited by 49 - Related articles - BL Direct - All 4 versions
MMMFQP He, AHGYK Csiszar, PA Li - 糖尿病学杂志, 2003 - journal.shouxi.net It is well known that diabetes aggravates brain damage in experimental and clinical stroke
subjects. Diabetes accelerates maturation of neuronal damage, increases infarct volume, and
induces postischemic seizures. The mechanism by which diabetes increases ischemic ... Related articles - Cached
PA Li, QP He, YB Ouyang, CL Liu, BR Hu, BK Siesjö - Brain research, 2001 - Elsevier The mechanisms underlying the aggravating effect of hyperglycemia on brain damage are still
elusive. The present study was designed to test our hypothesis that hyperglycemia-mediated
damage is caused by mitochondrial dysfunction with mitochondrial release of cytochrome ... Cited by 26 - Related articles - All 3 versions
J Dai, GFJM Vrensen, RO Schlingemann - Brain research, 2002 - Elsevier Diabetes-related cognitive dysfunction has been recognized for many years in humans, but the
pathogenesis of this condition is poorly understood. Evidence from animal studies suggests that
altered function of the blood–brain barrier (BBB) could be a potential cause contributing ... Cited by 16 - Related articles - All 4 versions
C Ding, QP He, PA Li - Experimental Neurology, 2004 - Elsevier Mitochondria play a critical role in the pathogenesis of cerebral ischemia. Acute hyperglycemia
has been shown to activate the mitochondria-initiated cell death pathway after an intermediate
period of ischemia. The objective of the present study was to determine if diabetic ... Cited by 17 - Related articles - All 4 versions
PA Li, L Gisselsson, J Keuker, J Vogel, ML Smith, W … - Brain research, 1998 - Elsevier Transient focal ischemia of brief duration (15–30 min) gives rise to brain damage. In normoglycemic
animals this damage usually consists of selective neuronal necrosis (SNN), and is largely confined
to the lateral caudoputamen. In hyperglycemic subjects damage occurs more rapidly, ... Cited by 31 - Related articles - All 4 versions
M Britton, J Rafols, S Alousi, JC Dunbar - Experimental Diabesity Research, 2003 - hindawi.com Experimental Diab. Res., 4:13–20, 2003 Copyright c 2003 Taylor & Francis 1543-8600/03 $12.00
+ .00 DOI: 10.1080/15438600390214581 ... The Effects of Middle Cerebral Artery Occlusion
on Central ... Mark Britton, Jose Rafols, Sarah Alousi, and Joseph C. Dunbar Cited by 7 - Related articles - View as HTML - All 10 versions
PA Li, I Rasquinha, QP He, BK Siesjö, K Csiszár, … - Journal of Cerebral …, 2001 - nature.com Previous histopathologic results have suggested that one mechanism whereby hyperglycemia
(HG) leads to exaggerated ischemic damage involves fragmentation of DNA. DNA fragmentation
in normoglycemia (NG) and HG rats subjected to 30 minutes of forebrain ischemia was ... Cited by 22 - Related articles - BL Direct - All 3 versions
DR Garris - Neuroscience letters, 1984 - ncbi.nlm.nih.gov The hypothalamic neurons of the diabetic Chinese hamster exhibited degradation associated
with the hyperglycemic, diabetic condition as compared with matched control animals. In
particular, neurons in the arcuate and ventromedial nuclei exhibited characteristic ... Cited by 3 - Related articles - All 2 versions
