Web Images Videos Maps News Shopping Gmail more »
Sign in
Scholar Home  
  Advanced Scholar Search
Scholar Preferences
Scholar Results 1 - 10 of about 101 related to Carpenter: PKCα is activated but not required during glucose-induced insulin secretion from.... (0.10 sec) 

PKCα is activated but not required during glucose-induced insulin secretion from rat …


L Carpenter, CJ Mitchell, ZZ Xu, P Poronnik, … - Diabetes, 2004 - Am Diabetes Assoc
The role of protein kinase C (PKC) in glucose-stimulated insulin secretion
(GSIS) is controversial. Using recombinant adenoviruses for overexpression of
PKCα and PKCδ, in both wild-type (WT) and kinase-dead (KD) forms, we here ...
Cited by 13 - Related articles - BL Direct - All 5 versions

Effects of protein kinase C inhibitors on insulin secretory responses from rodent pancreatic …


WS Zawalich, KC Zawalich - Molecular and Cellular Endocrinology, 2001 - Elsevier
The contribution of protein kinase C (PKC) to the regulation of insulin release
from perifused islets was explored using staurosporine or Gö 6976 to inhibit
the enzyme. Phorbol 12-myristate 13-acetate (PMA, 500 nM) addition to rat ...
Cited by 23 - Related articles - All 4 versions

Bimodal role of conventional protein kinase C in insulin secretion from rat pancreatic β cells

- nih.gov
H Zhang, M Nagasawa, S Yamada, H Mogami, … - The Journal of Physiology, 2004 - Physiological Soc
The present study was conducted to evaluate the role of conventional protein
kinase C (PKC) in calcium-evoked insulin secretion. In rat β cells transfected
with green fluorescent protein-tagged PKC-α (PKC-α–EGFP), a ...
Cited by 13 - Related articles - All 7 versions

Dynamics of Glucose-Induced Localization of PKC Isoenzymes in Pancreatic β-Cells

- diabetesjournals.org
N Warwar, S Efendic, CG Östenson, EP Haber … - Diabetes, 2006 - Am Diabetes Assoc
Glucose metabolism affects most major signal pathways in pancreatic β-cells.
Multiple protein kinases, including protein kinase C (PKC) isoenzymes, are
involved in these effects; however, their role is poorly defined. Moreover, ...
Cited by 19 - Related articles - BL Direct - All 5 versions

Chronic exposure to TPA depletes PKCalpha and augments Ca-dependent insulin secretion …

- physiology.org
WS Zawalich, M Bonnet-Eymard, KC … - American Journal of Physiology- Cell Physiology, 1998 - Am Physiological Soc
The insulin secretory responses of rat islets to glucose (15 mM),
12-O-tetradecanoylphorbol 13-acetate (TPA; 500 nM), and potassium (30 mM) were
determined from perifused islets cultured for 22-24 h in CMRL-1066 medium ( ...
Cited by 9 - Related articles - BL Direct - All 4 versions

Protein kinase C {delta} plays a non-redundant role in insulin secretion in pancreatic beta …

- jbc.org
T Uchida, N Iwashita, M Ohara-Imaizumi, T … - Journal of Biological Chemistry, 2007 - ASBMB
Protein kinase C (PKC) is considered to modulate glucose-stimulated insulin
secretion. Pancreatic cells express multiple isoforms of PKCs; however, the role
of each isoform in glucose-stimulated insulin secretion remains ...
Cited by 12 - Related articles - All 6 versions

Insulin secretion from islets of GK rats is not impaired after energy generating steps


N Katayama, SJ Hughes, SJ Persaud, PM … - Molecular and cellular endocrinology, 1995 - Elsevier
Insulin secretory responses of intact and electrically permeabilised islets of
Goto-Kakizaki (GK) rats, a novel model of non-insulin dependent diabetes
mellitus, and Wistar (control) rats were compared to investigate the ...
Cited by 8 - Related articles - All 5 versions

[CITATION] MECHANISMS OF SIGNAL TRANSDUCTION-Involvement of protein kinase C b2 in c-myc …


H Kaneto, K Suzuma, A Sharma, S Bonner- … - Journal of Biological Chemistry, 2002 - Baltimore [etc.]
Cited by 2 - Related articles

[CITATION] PI in't Veld, E. Maes, and M. Van De Winkel. 1982. Glucose-induced insulin release depends …


D Pipeleers - Proc. Natl. Acad. Sci. USA
Cited by 4 - Related articles

Ca 2+ handling of rat pancreatic β-cells exposed to ryanodine, caffeine, and glucagon


M Ahmed, E Grapengiesser - Endocrine, 2002 - Springer
Received November 2, 2001; Revised December 26, 2001; Accepted December 26,
2001. Author to whom all correspondence and reprint requests should be
addressed: Dr. Eva Grapengiesser, Department of Medical Cell Biology, ...
Cited by 4 - Related articles - BL Direct - All 4 versions


Result Page: 

1

2

3

4

5

6

7

8

9

10

Next


 


Go to Google Home - About Google - About Google Scholar

©2009 Google