- ►diabetesjournals.org D Accili - Diabetes, 2004 - Am Diabetes Assoc Type 2 diabetes arises from a combination of impaired insulin action and
defective pancreatic ß-cell function. Classically, the two abnormalities have
been viewed as distinct yet mutually detrimental processes. The combination ... Cited by 53 - Related articles - BL Direct - All 4 versions
D Accili, FCIDC Differentiation, SC as an … - Diabetes, 2004 - medscape.com Type 2 diabetes arises from a combination of impaired insulin action and
defective pancreatic β-cell function. Classically, the two abnormalities have
been viewed as distinct yet mutually detrimental processes. The combination ... Cited by 4 - Related articles - All 5 versions
D Accili - DIABETES, 2004 - Am Diabetes Assoc Type 2 diabetes arises from a combination of impaired insulin action and
defective pancreatic -cell function. Classically, the two abnormalities have
been viewed as distinct yet mutually detrimental processes. The combi- ... Related articles
FCIDC Differentiation, SC as an Opportunity - medscape.com Although it is clear that β-cells can proliferate in response to a host of
growth factors, the most parsimonious explanation for insulin resistance-induced
β-cell hyperplasia is that insulin itself promotes β-cell neogenesis and ... Related articles
SC as an Opportunity - medscape.com Insulin signaling can be viewed as a three-tiered system, in which a metabolic
response requires initially tyrosine phosphorylation, then phosphoinositol
generation, and ultimately serine/threonine phosphorylation. At each step ... Related articles
FCIDC Differentiation, SC as an Opportunity - medscape.com The theme of this review is to emphasize how a panoply of tissues contributes to
metabolic control. I have grouped them under the moniker of "the republic of
insulin resistance," and in doing so I probably overstepped the modesty of ... Related articles
FCIDC Differentiation, SC as an Opportunity - medscape.com The Controversial Role of Pancreatic Duct Cells in β-cell Neogenesis: The
just-published 2003 Lilly Lecture discusses how a panoply of tissues contributes ... Related articles
FCIDC Differentiation, SC as an Opportunity - medscape.com Signaling Pathways Regulating β-cell Function: The just-published 2003 Lilly Lecture
discusses how a panoply of tissues contributes to metabolic control.
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- ►nih.gov H Okamoto, J Nakae, T Kitamura, BC Park, I … - Journal of Clinical Investigation, 2004 - Am Soc Clin Investig The role of different tissues in insulin action and their contribution to the
pathogenesis of diabetes remain unclear. To examine this question, we have used
genetic reconstitution experiments in mice. Genetic ablation of insulin ... Cited by 42 - Related articles - BL Direct - All 9 versions
- ►jbc.org W Cao, QF Collins, TC Becker, J Robidoux, EG … - Journal of Biological Chemistry, 2005 - ASBMB Hepatic gluconeogenesis is essential for maintaining blood glucose levels during
fasting and is the major contributor to postprandial and fasting hyperglycemia
in diabetes. Gluconeogenesis is a classic cAMP/protein kinase A-dependent ... Cited by 39 - Related articles - BL Direct - All 5 versions
